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So you want to do a Gait Analysis: Part 4

This is the 4th in a multi part series. If you missed part 1, click here. For part 2, click here, part 3, click here

These are the basics, folks. We hope this is a review for many.

A quick review of the walking gait cycle components:

There are two phases of gait: stance and swing

Stance consists of:

  • Initial contact
  • Loading response
  • Midstance
  • Terminal stance
  • Pre-swing

Swing consists of:

  • initial (early) swing
  • mid swing
  • terminal (late) swing

today, lets explore Terminal stance

Terminal stance is one of the last stages of stance phase. Following midstance, where maximal pronation should be occurring, the stance phase foot should now begin supinating, initiated by the the opposite foot in swing phase moving forward of the center of gravity. 

Lets look at what is happening here at the major anatomical areas:

Foot

  • Supination begins from the opposite, swing phase leg (see above)
  •  the calcaneus inverts to neutral
  •  the center of gravity of the foot raises from its lowest point at midstance
  • The lower leg should begin externally rotating (as it follows the talus)
  • The thigh should follow the lower leg and should also be externally rotating; sometimes to a greater extent due to the shape and size of the medial condyle of the femur (which is larger than the lateral)
  • these actions are perpetuated by the gluteus maximus and posterior fibers of the gluteus medius, as well as posterior compartment of the lower leg including the flexor digitorum longus, flexor hallucis longus, peroneus longus and tibialis posterior
Ankle
  • The ankle should be 5 degrees dorsiflexed and in ankle rocker
  • the calcaneocuboid locking mechanism should be engaging to assist the peroneus longus in getting the head of the 1st metatarsal to the ground

Knee

  • near or at full extension. This is perpetuated by the quadriceps and biceps femoris, contracting concentrically and attenuated by the semi membranosis and tendonosis. The popliteus contracts eccentrically as soon as the knee passes midstance to keep the rates of external rotation of the tibia and femur in congruence.

Hip

  • The hip should be extending to 10 degrees.

Can you picture what is happening? Try and visualize these motions in your mind. Can you understand why you need to know what is going on at each phase to be able to identify problems? If you don’t know what normal looks like, you will have a tougher time figuring out what is abnormal.

Ivo and Shawn. Gait and foot geeks extraordinaire. Helping you to build a better foundation to put all this stuff you are learning on.

pictured used with permission from Foot Orthoses and Other Conservative Forms of Foot Care

Subtle Clues to Ankle Rocker Pathology: How good are your powers of observation ?

There are clues showing you there is motor pathology to ankle dorsiflexion, if you are paying close enough attention.

When we see motor pathology in ankle dorsiflexion we immediately begin to think about impairment to hip extension range of motion, gluteal strength, motor coordination and many other issues.

Here is a simple case. Observation skills are your greatest superpower when it comes to figuring out many gait and movement problems. But, you have to know what to look for and know what they mean before you can even hope to know how to fix things.
This is a simple video. It shows active ankle dorsiflexion in supination. We asked the client (a runner with right heel and persistent sesamoid pain following a healed sesamoid fracture) to perform simple ankle dorsiflexion. This is what we saw.

It should be clear to the observer that the end of the video shows attempted right dorsiflexion pulls the 2-5 toe extensors into the pattern quite aggressively and as a dominating faction. One can see toe abduction and extension with surprisingly little help from the long hallux toe extensor (EHL).  Dorsiflexion also fatigued early on the right. There is only one reason that the lesser toe extensors (EDL & EDB) are being over recruited, it’s because the EHL and tibialis anterior are weak and/or inhibited or have been pattern corrupted for one reason or another. Depending on this smallest of anterior compartment muscles over the EHL and tib anterior will mean that ankle rocker (dorsiflexion) is impaired. It also means that abnormal forefoot valgus posturing is expected (we could make a case for valgus or varus depending on other variables present). Passive ROM assessment confirmed the impaired ankle rocker with barely greater than 90 degrees ankle dorsiflexion ROM. This impairment will possibly do many things including:

  • premature heel rise
  • premature gastrocsoleus engagement
  • accentuated rear foot eversion (Rearfoot pronation)
  • midfoot pronation
  • strain of plantar fascia
  • premature forefoot loading response (strong clue for clients sesamoid fracture and persistent pain)
  • anterior/ posterior shin splints
  • hallux VALgus /bunion formation
  • long toe flexor dominance and many other things.

This clinical find plays nicely into the clients multiple symptoms (plantar pain and sesamoid problems) and functional gait pathology.
Restoring proper motor hierarchy and synchrony to the ankle dorsiflexion team (tib anterior, peroneus tertius, EDL, EHL) will reduce the need for solitary group overuse and impart forces where they should be when they need to be present. Impair the synchrony and problems ensue.

Help your client achieve the motion at the ankle mortise and they do not have to pass the buck into the foot.  Always test for skill, endurance and strength. Endurance is the most often forgotten assessment.  If endurance is lost early, the brain will begin to block out that end range of motion because it cannot be trusted, and thus posterior compartment tightness will be detected. This is an often common source of regional achilles and para-achilles tendonopathy. If your clients symptoms take time during activity to develop looking at the endurance of motor patterns may give the clue to your solution. 

Simple case, but you have to know your normal gait parameters, know functional anatomy and know how impaired mechanics factor into injury. 

Shawn and Ivo

The gait guys

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Spine pain and arm swing. Do you truly get this ? You had better.

We have all seen that runner who swings the one  arm more than the other, they may even violently thrust the one arm across the front of the torso. If you have been a spectator half way through any race you have seen this person. And, if you are watching carefully in your gym, lab, office or gait lab  you have seen the accentuated arm swing on one side (or is it the loss of arm swing on the opposite, we discussed some of these games in last weeks blog post here). You have also see the person who is running with a water bottle in their hand and altering their neurological arm-leg swing opposite pairing and thus their anti-phasic shoulder-pelvic girdle pairing (see attached photo). (If you are lost when we discuss the terms phasic and anti-phasic you will want to go and read this previous blog post.

Knowing that which you are seeing in your client is their highest level of neurologic motor compensation, and not likely their problem, represents a higher thought process in a diagnostician. Unfortunately, it also opens a whole bunch of clinical thought process mental gymnastics. 

Our purpose of today’s blog post is to revisit an important aspect of the clinical examination, observation.  Listening and watching (and knowing what you are seeing, and not seeing) are two of the biggest pieces of a clinical exam other than the hands on assessments. One has to be good at all of the pieces.  But then their is the knowledge base that is needed to base the information and choices upon so that the proper path to remedy can be chosen.  Without the knowledge the actions and choices can be dramatically incorrect and devastating to an athlete or client/patient.  Make the wrong choice for a patient and they do not get better, perhaps even get worse. Make the wrong choice for an athlete and you deepen their compensation and increase their risk for injury.  This is one of our pet peeves because we recognize that we have a deep knowledge base and yet we find ourselves without the certainty and answers on a regular basis and yet we see people making similar choices for clients and athlete with only a small piece of the knowledge necessary on their table to make those choices.  If you don’t know what you don’t know, and yet your still swimming in the risky waters, you are already in deep trouble. 

Here are two articles that you should be familiar with. We talk about them in depth in our “arm swing” online course #317 here.  These articles talk about phasic and antiphasic motions of the arms and shoulder-pelvic blocks.  They talk about spine pain and how spine pain clients reduce the antiphasic rotational (axial) nature of the shouder girdle and pelvic girdle. They elude to the subcortial pattern of choice to rotate them as a solid unit to reduce spine rotation, axial loading and compression and that spine pain disables the normal arm-leg pendulums.  If you do not know and  understand these principles, and you are training, treating or coaching people, you are a problem waiting to happen for your client. You, are the problem and your choices could likely hurt your client.  IF you do not know how to address them or fix them safely, it is your job to send them to someone who does. 

So the next time you see an aberrant arm swing, during your exam, your observations and your history better delve into all things relevant. How about that 20 year “healed” ankle fracture that your client dismisses as “oh, but that was 20 years ago, its not part of this problem i am having now”.  How about that episode of frozen shoulder that was “fixed” 15 years ago or that episode of hip or knee pain from falling on ice or the random big toe pain or the headaches ?  If they dismiss all of this because they are just coming to see you for spine pain or because their running partner says their arm swing stinks on the right you had better sit down for a longer ride, because you  know better now.  Unless you prefer to see life through tunnel vision. Sure it is easier, but don’t you want more for your client ?

Sorry for the rant.

Shawn and Ivo, …… the gait guys.

1. Eur Spine J. 2011 Mar;20(3):491-9. doi: 10.1007/s00586-010-1639-8. Epub 2010 Dec 24.
Gait adaptations in low back pain patients with lumbar disc herniation: trunk coordination and arm swing.  Huang YP et al.
2. J Biomech. 2012 Jan 10;45(2):342-7. doi: 10.1016/j.jbiomech.2011.10.024. Epub 2011 Nov 10.

Mechanical coupling between transverse plane pelvis and thorax rotations during gait is higher in people with low back pain.

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You can only “borrow” so much before you need to “pay it back”

How can feet relate to golf swing?

This 52 year old right handed gentleman presented with pain at the thoracolumbar junction after playing golf. He noticed he had a limited amount of “back swing” and pain at the end of his “follow through”.

Take a look a these pix and think about why.

Hopefully, in addition to he having hairy and scarred legs (he is a contractor by trade), you noted the following

  • Top left: note the normal internal rotation of the right hip; You need 4 degrees to walk normally and most folks have close to 40 degrees. He also has internal tibial torsion.
  • Top right: loss of external rotation of the right hip. Again, you need 4 degrees (from neutral) of external rotation of the hip to supinate and walk normally.
  • Top center:normal internal rotation of the left hip; internal tibial torsion
  • 3rd photo down: limited external rotation of the left hip, especially with respect ti the amount of internal rotation present; this is to a greater degree than the right
  • 4th and 5th photos down: note the amount of tibial varum and tibial torsion. Yes, with this much varum, he has a forefoot varus.

The brain is wired so that it will (generally) not allow you to walk with your toes pointing in (pigeon toed), so you rotate them out to somewhat of a normal progression angle (for more on progression angles, click here). If you have internal tibial torsion, this places the knees outside the saggital plane. (For more on tibial torsion, click here.) If you rotate your extremity outward, and already have a limited amount of range of motion available, you will take up some of that range of motion, making less available for normal physiological function. If the motion cannot occur at the knee or hip, it will usually occur at the next available joint cephalad, in this case the spine.

The lumbar spine has a limited amount of rotation available, ranging from 1.2-1.7 degrees per segment in a normal spine (1). This is generally less in degenerative conditions (2).

Place your feet on the ground with your feet pointing straight ahead. Now simulate a right handed golf swing, bending slightly at the waist and  rotating your body backward to the right. Now slowly swing and follow through from right to left. Note what happens to your hips: as you wind back to the right, the left hip is externally rotating and the right hip is internally rotating. As you follow through to the left, your right, your hip must externally rotate and your left hip must externally rotate. Can you see how his left hip is inhibiting his back swing and his right hip is limiting  his follow through? Can you see that because of his internal tibial torsion, he has already “used up” some of his external rotation range of motion?

If he does not have enough range of motion in the hip, where will it come from?

he will “borrow it” from a joint more north of the hip, in this case, his spine. More motion will occur at the thoracolumbar junction, since most likely (because of degenerative change) the most is available there; but you can only “borrow” so much before you need to “Pay it back”. In this case, he over rotated and injured the joint.

What did we do?

  • we treated the injured joint locally, with manipulation of the pathomechanical segments
  • we reduced inflammation and muscle spasm with acupuncture
  • we gave him some lumbar and throacolumbar stabilization exercises: founders exercise, extension holds, non tripod, cross crawl, pull ups
  • we gave him foot exercises to reduce his forefoot varus: tripod standing, EHB, lift-spread-reach
  • we had him externally rotate both feet (duck) when playing golf

The Gait Guys. Helping you to store up lots “in your bank” of foot and gait literacy, so you can help people when they need to “pay it back”, one case at a time.

(1) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2223353/

(2) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3705911/

Podcast 68: Gait , Arm Swing, Neuro-developmental Windows

A. Link to our server:

Direct Download: http://traffic.libsyn.com/thegaitguys/pod_68ffinal.mp3

Permalink: http://thegaitguys.libsyn.com/podcast-68

B. iTunes link:

https://itunes.apple.com/us/podcast/the-gait-guys-podcast/id559864138

C. Gait Guys online /download store (National Shoe Fit Certification and more !) :

http://store.payloadz.com/results/results.aspx?m=80204

D. other web based Gait Guys lectures:

www.onlinece.com   type in Dr. Waerlop or Dr. Allen,  ”Biomechanics”

______________

Today’s Show notes:

1.Brain implant helps paralysed man move his hand
http://realitysandwich.com/220399/brain-implant-helps-paralysed-man-move-his-hand-wired-uk/?u=95820

2. Has Science Finally Confirmed the Existence of Acupuncture Points, Validating Chinese Medicine?

3.This Tiny, Whip-Tailed Robot Can Administer Meds Anywhere In the Body
4. It matters what you put on your kids feet
“Shoes affect the gait of children. With shoes, children walk faster by…
5. Normal gait development.
6. Myelination
7. Arm swing in kids.
8. Arm swing and gait speed.
Arm Swing Truths, and Lies.
We have been reminded over and over again in recent weeks how intimately arm swing is tied to leg swing. We have recently had clients in our practices with strokes (ischemic and hemorrhagic), transverse myelitis, inflamma…

Arm Swing Truths, and Lies.

We have been reminded over and over again in recent weeks how intimately arm swing is tied to leg swing. We have recently had clients in our practices with strokes (ischemic and hemorrhagic), transverse myelitis, inflammatory neurologic disorder and the plethora of biomechanically pain-mediated gait responses affecting the limbs, including the upper limbs which manifest many variations in these people’s normal gait neuro-mechanics.

We are even reminded of the recent triathlete who had a right hip weakness that was allowing him to drift into the right frontal plane in running and biking. Upon asking about further symptoms he mentioned left hand tingling on longer bike training rides.  We asked if he recalled sliding/shifting onto the right side of the saddle/seat often to find power in the right leg and he mentioned matter of fact that it is a constant awareness. We then suggested that he was having to over pressure into the left handle grips to keep the bike on a straight line because of the right pelvis-saddle shift. He was wide eyed and shocked that it was what he was in fact aware of.  Moral of the story: even in on the bike opposite arm and leg action are intimately tied together.  After testing and assessment it was clear that a function TOS (thoracic outlet syndrome) was in effect because of shortness and increased tone in the left pectoral chest wall compromising neurovascular bundle compression and generating hand paresthesias (numbness/tingling).  A simple fix if you fix the right pelvic frontal plane drift.  If you try to fix the TOS at the shoulder-neck level resistance to progress is likely.

Arm swing is a sneaky thing. There are many variables. We have discussed many of these arm swing variables in 38 previous blog posts (link here) and we have whole lecture here on arm swing (this slide is part of that in depth lecture).

In the pubmed article below there is new research delving into passive and active components of arm swing. There are both, clearly. But what a reader needs to take away is that a clinical examination must be part of every assessment to discover the active components (muscular and neuromuscular) that are missing (ie. weak posterior deltoid, lat dorsi etc) and which need fixing and rehabilitation and the passive components that are inhibitory to the big picture (mobility, stability).

It should be clear by now, if you have been with us for at least the last year, that what you see in someone’s gait is their compensation, not their problem. Addressing resolution measures to change what you see is a path to deepening the compensation or developing others.  Arm swing is intimately tied to the lower limbs, and powerfully so to the opposite leg. A deficit in the leg will be expressed in some way in the opposite upper limb, which in turn forces a compensatory change in the opposite upper limb and thus down into the “other” opposite lower limb.  One thing affects many. The wrong intervention drives bigger problems, so make sure you know your gait “normal” parameters and be sure a clinical examination is a huge part of your discovery toward the answers for your client.

Shawn and Ivo … . .  the gait guys

Pubmed abstract link: http://www.ncbi.nlm.nih.gov/pubmed/24865637

Gait Posture. 2014 Jun;40(2):321-6. Epub 2014 May 6.

Arm swing in human walking: What is their drive?

Abstract

The results confirm that passive dynamics are partly responsible for arm swing during walking. However, without muscle activity, passive swing amplitude and relative phase decrease significantly (both p<0.05), the latter inducing a more in-phase swing pattern of the arms. Therefore, we conclude that muscle activity is needed to increase arm swing amplitude and modify relative phase during human walking to obtain an out-phase movement relative to the legs.

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Flat Dogs

Take a look at these pedographs. Wow!

  • No rear foot heel teardrop.
  • No midfoot arch on left foot and minimal on right.
  • An elongated 2nd metatarsal bilaterally and forces NOT getting to the base of the 1st metatarsal and stalling on the 2nd: classic sign of an uncompensated forefoot varus.
  • increased printing of the lateral foot on the right

Knowing what you know about pronation (need a review? click here) Do you think this foot is a good lever? Do you think they will be able to push off well?

What can we do?

  • foot exercises to build the intrinsic and extrinsic muscles of the foot (click here, here, here, and here for a few to get you started)
  • perhaps an orthotic to assist in decreasing the pronation while they are strengthening their foot
  • motion control shoe? Especially in the beginning as they are strengthening their feet and they fatigue rather easily

The prints do not lie. They tell the true story of how the forces are being transmitted through the foot. For more pedograph cases, click here.

The Gait Guys. Teaching you more about the feet and gait. Spreading gait literacy throughout the net! Do your part by forwarding this post to someone who needs to read it.

The weeping calf and the deconstructed arm swing.

Last week we showed you this video and blog post of a compressive left lower leg neuropathy and what it looks like when both heel and toe walking are attempted when both are compromised. It was nothing exciting but to see both in a clinical presentation is not all that common.

In today’s videos (the one above and this one here), the videos were all shot on the same day incidentally, we wanted you to see this gentleman’s gait in it’s normal gait pattern attempt.  Because less of the extremes of range and strength are required, it is far more difficult to detect the issues than in last week’s video clip (here).

There are plenty of things to talk about in this video but lets just point out one of them here today.  Remember, the lesion is in the left lower leg.

Absent right arm swing. 

We have been harping about arm swing for a long time.  Go to the search box here on our blog and type in “arm swing” and you will find an abundance of articles on the biomechanics and neurology of arm swing and how it is tied to leg swing.  In this case we have foot drop and impaired calf raise (video link) on the left. Their function is impaired/depressed. We are seeing this matching in the absence of right upper limb swing.  Remember, most of the time the upper limb takes the queue from the opposite lower limb. This is why coaching arm swing changes is not a sound idea most of the time, look for functional opportunities for changes in the opposite lower limb if deficits are present there.  

Part of what you are seeing is the increased activity in the left arm swing.  Why ? Because the client is abruptly lurching off of the left leg because of the stability and strength deficits in that limb. The brain knows that bearing weight on the left limb has challenges.  This causes an abrupt pitch (early departure) forward onto the right leg and this will be met with increased left arm swing (go limb around your home or office, you will see that it is a coupled phenomenon).  So, is it increased left arm swing you are seeing because of this issue we just mentioned or are you seeing decreased right arm swing because of the matching neuro-suppression of left leg ? 

This is where your clinical examination must come into play. Shame on anyone that is making the changes without clinical information. One must see that there rare two (at least) possible scenarios for the differential in arm swing. And one must also see that the arms in this case are not the issue, that it is the left lower limb deficits that are driving the issue.  Guaranteed.

Arm swing……..more to it than you might think.

Shawn and Ivo, The gait guys

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So a patient presents to your office with a recent history of a L total knee replacement 8 weeks ago AND a recent history of a resurgence of low back pain, supra iliac area on the L side. Hmmmm. Hope the flags went up for you too!

His global lumbar ROM’s were 70/90 flexion with low back discomfort at the lumbo sacral junction, 20/30 extension with lumbosacral discomfort, left lateral bending 10 degrees with increased pain (reproduction); right lateral bending 20 degrees with a pulling sensation on the right. Extension and axial compression of the lumbar spine in left lateral bending reproduced his pain.

Neurologically he had an absent patellar reflex on the left, with diminished sensation over the knee medially and laterally. Muscle strength 5/5 in LE; sl impaired balance in Left single leg standing. There was incomplete extension of the left knee, being at 5 degrees flexion (right side was zero).

He has a right sided leg length deficiency (or a left sided excess!) of 5 mm. Take a look at the tibial lengths in the 1st 3 pictures. See how the left is longer? In the next shot, do you see how the knee cannot completely extend? Can you imagine that the discrepancy would probably be larger if it did?

Now look at the x rays. We drew a line across from the non surgical leg to make things clearer.

Now, think about the mechanics of a longer leg. That leg will usually pronate more in an attempt to shorten the leg, and the opposite side will supinate to attempt to lengthen. Can you see how this would cause clockwise pelvic rotation (in addition to anterior pelvic rotation)? Can you see this patients in the view of the knees from the top? Do you understand that the lumbar spine has very limited rotation (about 5-10 degrees, with more movement superiorly (1)  ). Does it make sense that the increased range of motion could effect the disc and facet joints and increase the patients low back pain?

So, how do we fix it? Have you seen the movie “Gattica”? Hmmm….A bit extreme. How about a full length 3mm sole lift to start, along with specific joint manipulation to restore normal motion and some acupuncture to reduce inflammation? We say that is a good start.

The Gait Guys. Increasing your gait literacy with each and every post. If you liked this post, please send it to someone else for them to enjoy and learn. 

(1) Three-Dimensional In Vivo Measurement of Lumbar Spine Segmental Motion Ruth S. Ochia, PhD, Nozomu Inoue, MD, PhD, Susan M. Renner, MS, Eric P. Lorenz, MS, Tae-Hong Lim, PhD, Gunnar B. Andersson, J. MD, PhD, Howard S. An, MD Spine. 2006;31(15):2073-2078.

Podcast 67: Biotech of Running's Future, Rothbart's Foot, 100 Ups

A. Link to our server:

Direct Download:

http://traffic.libsyn.com/thegaitguys/pod_67finals.mp3

Permalink: http://thegaitguys.libsyn.com/podcast-67

B. iTunes link:

https://itunes.apple.com/us/podcast/the-gait-guys-podcast/id559864138

C. Gait Guys online /download store (National Shoe Fit Certification and more !) :

http://store.payloadz.com/results/results.aspx?m=80204

D. other web based Gait Guys lectures:

www.onlinece.com   type in Dr. Waerlop or Dr. Allen,  ”Biomechanics”

______________

Today’s Show notes:

The cyborg era begins next week at the World Cup
 
The One Exercise That Just Might Change Your Running Forever
 

What Foot Strike Photos From 10K Olympic Trials Say About Barefoot Running by 

A Serious Gait Problem: Pancompartmental Compromise of the Lower Leg.

“Pan” is a prefix (combining form) meaning all, entire, everything, everywhere 

This was a case we discussed during a more recent podcast, perhaps pod 63 or 64? This doctor had fallen asleep with the left leg dangling over the side of his bed. The issue was that the leg not only dangled over the mattress, but also over a wooded side bed frame, so there was a firm upward compression into the posterior/popliteal compartment. He awoke the next day with complete loss of function of the foot and ankle.  This video is 8 weeks after the compressive event and there has been a significant improvement in function, but there are still some deficits here.  Can you see them ?  We will show you come other video clips in a future blog post discussing some other components of his gait but lets get you familiar with the case today.

What you should see here:

1- Left heel shows a staggered drop. He cannot hold heel rise because of compromise to the posterior compartment strength (gastrocsoleus complex). This was a drastic improvement from his complete inability to heel rise at all at on his initial visit. You can easily see the fatiguability of the calf after just a few steps. 

2- There is a pathetic attempt at heel walking; gross function testing of the anterior compartment. What appears to be an attempt at just right heel walking is actually an attempt to do it on both sides, there is just still so much weakness in the left anterior compartment that you cannot even see his attempts to dorsiflex the foot/ankle or toes. But, what we do not show here is that he has non-weight bearing dorsiflexion now, which was completely absent for the first 6 weeks.  

Neuronal regeneration is possible. It takes time.  Depending on your referenced source the numbers vary. But in his case, in 8 weeks there is progressive improvements and he can say for certain that in the last 2 weeks they are exponential.  The time to restoration of neuronal function is said to be directly proportional to the measurable length of nerve damage.  

What is interesting in this case, is that there is anterior and posterior compartment neurologic compromise. This was a case of vascular and mechanical compression to the neurovascular bundle at the popliteal/knee level. 

Wallerian degeneration is a process that results when a nerve is severely damaged. The axon of the nerve which is separated from the neuron cell body degenerates distal to the injury. The part of the axon distal to the injury begins its degeneration within 24-36 hours of the lesioning event and is followed by myelin sheath degradation. Somewhere around 4 days from the time of the injury, the distal end of the portion of the nerve fiber proximal to the lesion begins sprouting in an attempt to regrow and fill the gap along the length of axonal damage. Sources vary, but many seem to indicate a 1mm per day reinnervation. 

More on this case next time, but the stage has been set.

Shawn and Ivo

Another IFGEC Certified Doc!
Here is what Dr Brad Hochstein has to say about the National Shoe Fit Certification Program.
&ldquo;Taking the National Shoe Fit Certification course has introduced me to many things that I didn’t take into consideration…

Another IFGEC Certified Doc!

Here is what Dr Brad Hochstein has to say about the National Shoe Fit Certification Program.

“Taking the National Shoe Fit Certification course has introduced me to many things that I didn’t take into consideration when working with my patients. The depth of information introduced is very helpful and presented more clearly than other courses I have taken in the past. I have studied a lot of the concepts that are introduced through the program in the past but struggled to put everything together. This certification did just that for me. It has helped me to link things together and look “outside the box” more than I had in the past.


I am a chiropractor with an extensive functional/biomechanics background and this has added another level of depth to my practice assessment and treatment. I am excited to incorporate the knowledge I have gained through this certification into my practice to give my patients an even better experience!”

Is it time to up your knowledge base and separate yourself from other clinicians, coaches, trainers, therapists and retailers?

Want to know more? Email us at : thegaitguys@gmail.com

Can you see it?
Here we are again. We have looked at this picture before; once about head tilt, and another about flip flops and form.
Take a good look at this picture and what is different about the child in blue all the way to the right and all th…

Can you see it?

Here we are again. We have looked at this picture before; once about head tilt, and another about flip flops and form.

Take a good look at this picture and what is different about the child in blue all the way to the right and all the others with the exception of the boy in pink, that we really cannot see?

Can you see it? No, we don’t mean the flip flops (but if you caught that all the boys were in sneakers and all the girls are in flip flops, you are good!)

How about looking at arm swing? Remember this post on arm swing and crossover gait, with the simple cue for correction? All of the children EXCEPT the boy in blue, are drawing their arms ACROSS their body (ie: flexion, internal rotation and adduction). Take a look at their legs. Yep, crossover gait (flexion, internal rotation and adduction). Little boy blues arms are going relatively straight and going in the saggital plane, where the others are going in the coronal plane.

We are not saying that blue does not have some gait challenges, like his torso shift to the left (or pelvic drift to the right), most likely do to gluteus medius weakness or inappropriate firing of the gluteus medius on the left stance phase leg; or his head tilt to the right, which most likely represents a compensation for the right pelvic drift and left body lean.

Arm swing. A very important clue to the puzzle we call gait and compensation. It is more prevalent than you think, and, in some cases, easily corrected with a simple cue.

The Gait Guys. Making it real and pertinent, in each and every post.

Podcast 66: Stem Cells, Running Form, Dartfish & Case Studes

A. Link to our server:

Direct Download: http://traffic.libsyn.com/thegaitguys/pod_66final.mp3

Permalink: http://thegaitguys.libsyn.com/podcast-66-stem-cells-running-form-dartfish-case-studes-0

B. iTunes link:

https://itunes.apple.com/us/podcast/the-gait-guys-podcast/id559864138

C. Gait Guys online /download store (National Shoe Fit Certification and more !) :

http://store.payloadz.com/results/results.aspx?m=80204

D. other web based Gait Guys lectures:

www.onlinece.com   type in Dr. Waerlop or Dr. Allen,  ”Biomechanics”

______________

Today’s Show notes:

Duke researchers have found a new type of neuron in the adult brain that is capable of telling stem cells to make more new neurons.

 
2. A closer look at iOS 8′s Health app (video)http://9to5mac.com/2014/06/02/a-closer-look-at-ios-8s-health-app-video/
new HealthKit platform aggregator will allow developers of various health and fitness apps to have all related data populate within the Health app in iOS 8.
 
3. something fun bc it is from the onion……but there is some truth to the placebo right ?
American Medical Association Introduces New Highly Effective Placebo Doctors
American Medical Association announced Thursday the introduction of new placebo doctors to administer general practice medical care to the American public. 
4. a beautiful example from our blog post today June 4th on what you see isnt always the problem
The Right Form For Running - Dartfish
“The video showed that his right foot was … .
 
some random talk we can do on asymetries and symmetry– 
 
6. Case studies on posture, pronation, osteitis pubis and more.

Correcting a cross over gait with arm swing? Is it really THAT easy? Sometimes, yes!

We noticed this patient had a cross over gait while running (1st few seconds of video. need to know more about crossover gait? click here). We noted she was crossing her arms over her body as well. We than had her run her hands and arms straight out. See the crossover disappear? Need to know more about arm swing? click here

We the had her do the same while walking. Easier to see, eh? That’s because it is often easier to “fudge” things when you are moving faster (ie: the basal ganglia of nervous system can interpolate where the body part is supposed to be, and because of momentum, there is less need for precision). When we do things slowly (like the 3 second Test), more precision is needed. Watch this short video clip a few more times.

The arms are essentially adducting when the arms cross over. The arms are reciprocally paired with the contralateral lower extremity. When you make a change in one, you often will make a change in the other.

Subtle. Yes. Easier to see when the task becomes more difficult. Yes. Pay attention, the answer is often right there if you look closely enough.

Providing the clues to help you be smarter, better, faster, stronger; we are The Gait Guys

special thanks to “Q” for allowing us to publish this video : )

The 5 Point Turn (in a human).  Do you know this gait problem ?

Here is a video link for the full video case study with diagnosis and more details on this client’s gait but our point here today is to look at the uniquely pathologic turning motor pattern deployed by this patient.

Gait analysis is so much more than watching someone move on a treadmill. Forward momentum at a normal speed can blur out many of a person’s gait pathologies.  We discussed this in detail in this blog post on slowing things down with the “3 second gait challenge”.  Furthermore, most gait analysis assessments do not start seated, then watching the client progress to standing, and then initiating movement.  Watching these intervals can show things that simple “gait analysis” will not.  Finding stability over one’s feet and then initiating forward motion can be a problem for many.  Those first moments after attaining the standing position afford momentum to carry the person sideways just as easily as carrying them forward. In other words, once momentum forward begins, a normal paced gait can make it difficult to see frontal plane deficits.  Our point here, transitional movements can show clues to gait problems and turning to change direction is no different.

Typically when we turn we use a classic “plant and pivot” strategy.  We step forward on a foot (right foot for example here), transfer a majority load on that forward right foot, we then pivot the left foot in the next anticipated direction of movement, and then push off the right foot directionally while spinning our body mass onto that left foot before initiating the right limb swing through to continue in the new direction.  This is not what this patient does. Go ahead, stand up and feel these transitions, if you are healthy and normal they are subconscious weight bearing transitions but for some one who is old and losing strength and proprioception/balance or some one with neurologic decline for one reason or another, these directional changes can be extremely difficult as you see in this video here. A full 180 degree progression is often the most difficult when things get really bad.  And more so, if one leg is more compromised than the other, turning one way a quarter turn (a 90 degree directional change) might be met with an alternative 270 degree multiple-point turn in the opposite direction over the more trusted limb to get to the same directional change. When there is posterior column disease or damage this seemingly simple “plant/weight shift/ pivot and push off” cannot be trusted. So a 5 point (or more) turn is deployed to be sure that small choppy steps maximize minimal loss of feel and maximal ground contact feel. This can be seen clearly in this video above.

Full video case link here:https://www.youtube.com/watch?v=AYmzQL_NSeI

Just some more things to think about in  your gait education.  Watch your clients move from sit to stand, from stand to initiating gait, and then watch closely their turning strategies. At the very least, have them make several passes making their about-face turns both to the right and the left. You will often see a difference.  Watch for unsteadiness, arm swing changes, cross over steps, reaching for stability (walls, furniture etc), moving of the arms into abduction for a ballast effect and the like. Then correlate your examination findings to your gait analysis.  Then, intervene with treatment and rehab, and review their gait again. Remember, explaining their deficiencies is a huge part of the learning process. Make them aware of their 5 point turns, troubles pivoting to the right or the left, and make them understand why they are doing the goofy one-sided rehab exercises. Understanding what is wrong is a huge part of fixing your client’s problems.

* Remember: if your client is having troubles on a stable surface (ie. the ground) then they should engage some rehab challenges on the ground. Giving them a tilt board or bosu or foam pad (ie. making the ground more unstable) will make things near impossible.  This is not a logical progression, we like to say, “if you can’t juggle one chainsaw we won’t give you 3”. Improve their function on a stable surface first, then once improvements are seen, then progress them to unstable surfaces.  

Shawn and Ivo

The gait guys.  

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Spanking the orthotic: The effects of hallux limitus on the foot’s longitudinal arch.

But the issues do not stop at the arch. If you have been with us long enough, you will have read about the effects of the anterior compartment (namely the tibialis anterior, extensor digitorum and hallucis and peroneus tertius muscles) strength and endurance on the arch.

Here we have a very troubled foot. This foot has undergone numerous procedures, sadly. Today we will not talk about the hallux varus you see here, a virtual unicorn in practice  (and acquired in this case) nor do we want to discuss the phalangeal varus drift. We want to draw your attention to the obvious impairment of the 1st MTP (metatarsophalangeal joint) dorsiflexion range.  You can see the large dorsal crown of osteophytes, a dorsal buttress to any hallux dorsiflexion.  There is under 10 degrees of dorsiflexion here, not even enough worth mentioning.  We have said it many times before, if you lose a range at one joint usually that range has to be accommodated for proximal or distal to the impaired joint. This is a compensation pattern and you can see it here in the hallux joints themselves.

Here you can see that some of the dorsiflexion range has been acquired in the proximal phalangeal joint.  We like to call this “banana toe” when explaining it to patients, it is a highly technical term but you are welcome to borrow it. This occurred because the joint was constantly seeing the limitation of dorsiflexion of the 1st MTP joint and seeing, and accommodating to, the demands of the need for more dorsiflexion at toe off. 

But, here is the kicker. You have likely seen this video of ours on Youtube on how to acquire a foot tripod from using the toe extensors to raise the arch.  Video link here  and here.  Well, in his patient’s case today, they have a limitation of 1st MTP dorsiflexion, so the ability to maximally raise the arch is impaired. The Windlass mechanism is broken; “winding” of the plantar fascia around the !st MTP mechanism is not sufficiently present. Any limitations in toe extension (ie dorsiflexion) or ankle dorsiflexion will mean that :

1. compensations will need to occur

2. The Windlass mechanism is insufficient

3. gait is impaired at distal swing phase and toe off phases

4. the anterior compartment competence will drop (Skill, endurance, strength) and thus injury can be more easily brought to the table.

In this patient’s case, they came in complaining of burning at the top of the foot and stiffness in the anterior ankle mortise area.  This would only come on after a long brisk walk.  If the walk was brisk yet short, no problems. If the walk was long and slow, no problems.  They clearly had an endurance problem and an endurance challenge in the office showed an immediate failure in under 30 seconds (we will try to shoot a quick video so show our little assessment so be patient with us). The point here today is that if there is a joint limitation, there will be a limitation in skill, strength or endurance and very likely a combination of the 3. If you cannot get to a range, then any skill, endurance or strength beyond that limitation will be lost and require a compensation pattern to occur.  This patient’s arch cannot be restored via the methods we describe here on our blog and it cannot be restored by an orthotic. The orthotic will likely further change, likely in a negative manner, the already limited function of the 1st MPJ. In other words, if you raise the arch, you will shorten the plantar fascia and draw the 1st MET  head towards the heel (part of the function of the Windlass mechanism) and by doing this you will plantarflex the big toe … .  but weren’t we praying for an increase in dorsiflexion of the limitus big toe ? ……..yes, exactly !  So use your head  (and spank the orthotic when you see it used in this manner.  ”Bad orthotic, bad orthotic !”)

So think of all of this the next time you see a turf toe / hallux rigidus/ hallux limitus. Rattles your brain huh !?

This is not stuff for the feint of heart. You gotta know your biomechanics.

Shawn and Ivo … .the gait guys

Addendum for clarity:

a Facebook reader asked a question:

From your post: “if you raise the arch, you will shorten the plantar fascia and draw the 1st MET head towards the heel (part of the function of the Windlass mechanism) and by doing this you will plantarflex the big toe … . but weren’t we praying for an increase in dorsiflexion of the limitus big toe ? ” I always thought when the plantar fascia is shortened, it plantar flexes the 1st metatarsal (1st ray) and extends (dorsiflexes) the 1st MTP joint….

Our response:  

We should have been more clear, our apologies dear reader.  Here is what we should have said , ” The plantar fascia is non-contractile, so it does not shorten. We meant conceptually shorten. When in late stance phase, particularly at toe off when the heel has raised and forefoot loading is occurring, the Windlass mechanism around the 1st MET head (as the hallux is dorsiflexing) is drawing the foot into supination and thus the heel towards the forefoot (ie passive arch lift). This action is driving the 1st MET into plantarflexion in the NORMAL foot.  This will NORMALLy help with increasing hallux dorsiflexion. In this case above, there is a rigid 1st MTP  joint.  So this mechanism cannot occur at all. In this case the plantar fascia will over time retract to the only length it does experience. So, if an orthotic is used, it will press up into the fascia and also plantarflex the 1st MET, which will carry the rigid toe into plantar flexion with it, IN THIS CASE.”

What&rsquo;s up, Doc?
Nothing like a little Monday morning brain stretching and a little Pedograph action.
This person had 2nd metatarsal head pain on the left. Can you figure out why?
Let&rsquo;s start at the rear foot:
limited calcaneal eversion (…

What’s up, Doc?

Nothing like a little Monday morning brain stretching and a little Pedograph action.

This person had 2nd metatarsal head pain on the left. Can you figure out why?

Let’s start at the rear foot:

  • limited calcaneal eversion (pronation) L > R. The teardrop shape is more rounded on the left. This indicates some rigidity here.
  • note the increased pressure at the  medial calcaneal facets on each side with the increased printing
  • very little fat pad displacement overall

Now let’s look at the mid foot:

  • decreased mid foot pronation on the L. See how thin the line is going from the rear foot to the forefoot along the lateral column? This indicates a high lateral longitudinal arch

Now how about the fore foot?

  • increased printing under the met heads bilaterally; L >> R
  • increased printing of 1st met head L >> R
  • increased printing at medial proximal phalynx of hallux  L >> R
  • increased printing of distal phalanges of all toes L >> R

 Figure it out?

What would cause increased supination on the L?

  • short leg on L
  • more rigid foot on L
  • increased pronation on the R

Did you notice the elongated 2nd metatarsals (ie: Morton’s toe) on each foot?

Here is what is going on:

  • there is no appreciable leg length deformity, functional or anatomical
  • The Left foot is more rigid than the Right, thus less rear, mid and fore foot pronation, thus it is in relative supination compared to the right foot

do this: stand and make your L foot more rigid than the right; take a step forward with your right foot, what do you notice?

  • Can you feel how when your foot is supinated
  • can you see how difficult it is to have ankle rocker at this point? remember: supination is plantar flexion, inversion and adduction
  • Can you feel the weight of the body shift to the outside of the foot and your toes curl to make the foot more stable, so you do not tip to the left?
  • now, how are you going to get your center of mass forward from here? You need to press off from your big toe (hallux)

Wow, does that make sense now?

What’s the fix?

  • create a more supple foot with manipulation, massage, muscle work
  • increase ankle rocker by training the anterior compartment (shuffle walks, lift/spread/reach exercise, heel walking, Texas walk exercise, etc)
  • have them walk with their toes slightly elevated
  • we are sure you can think of more ways as well!

The Gait Guys. Increasing your gait literacy with each and every post. If you liked this post, tell others and spread the word. If you didn’t like this post, tell us! We value your constructive feedback.