When we try to dorsiflex through the midfoot instead of the ankle.

A foot bump. Read on . . .

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We see this kind of thing all the time. This is a fixed pes planus (flat foot). When we dorsiflex the big toe, the arch does not go up as you see in the photo. That is passive dorsiflexion, if the arch does not go up passively, there is no way you are actively going to achieve this. And, using an orthotic to "attempt" to raise this arch is not only pointless, but it is futile and it will likely cause them pain. This arch does not rise, no matter how hard you put up into it. The bump, that is the navicular bone, and its associated arthritic build up at the adjacent joints, and likely soft tissue accommodation/hypertrophy. You can't needle, ultrasound, tape, adjust or rub this bump away, so stop wasting your and your patient's time selling them that wasteful thinking. It ain't gonna happen.
This is what happens when someone earns a collapsed longitidinal arch, the 1st metatarsal no longer plantarflexes (arch up) and it becomes fixed in dorsiflexion, thus affecting the mechanics at the proximal aspect of the 1st ray complex (navicular-cuneiform-met intervals).
Why? This happened because this client has significantly compromised ankle mortise dorsiflexion, and they chose to find it at the next joint complex distally, as mentioned above. So, they are finding pseudo-ankle rocker at arch collapse? Yes, we discuss this often, more pronation will advance the tibia forward. It is not desirable, but moving forward has to occur, and some people have no choice but to find it from excessive internal rotation and pronation of the limb. And this is what happens when it happens over years. Now the deformity is painful itself in the shoe, it is a new set of problems for this client.
Can this problem occur in reverse ? Yes, a loss of hallux dorsiflexion can afford the same end result.
We have a rule, at the very VERY least, check the joint above and below the area of problem/symptom. Often you will find another piece of the puzzle causing your client's pain.

Right arch pain, can you see a possible reason in this video?

Do you see a possible reason for right foot pain? There is something not kosher to be seen. It doesnt mean it is valid, or the cause, or that it is primary or secondary, but it should be something that cues up a clinical exam focus to rule in/rule out.
Answer below (don;'t read further, test yourself)
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the right hallux does not fully extend. And we know that hallux dorisflexion at the 1st MTP joint engages the windlass, and helps to plantarflex the 1st MET and raise the arch and prepare the foot for loading and for forefoot transition. If the hallux doesn't extend sufficiently (like in a hallux rigidus, painful turf toe etc) then we can have some loading issues. Just something to think about. In this case, it was the cause and answer. But might not always be such.

Podcast 106: Understanding Tendonopathies & Asymmetrical Bone density in athletes.

* Plus the global effects of Hallux Limitus, & Chronic exposure to routine high-impact, gravitational loads afforded to the support limb preferentially improved bone mass and structure

Show Sponsors:

newbalancechicago.com
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Other Gait Guys stuff

2 Podcast links: 

http://traffic.libsyn.com/thegaitguys/pod_106f.mp3

http://thegaitguys.libsyn.com/episode-106

B. iTunes link:
https://itunes.apple.com/us/podcast/the-gait-guys-podcast/id559864138

C. Gait Guys online /download store (National Shoe Fit Certification & more !)
http://store.payloadz.com/results/results.aspx?m=80204

D. other web based Gait Guys lectures:
Monthly lectures at : www.onlinece.com type in Dr. Waerlop or Dr. Allen, ”Biomechanics”

-Our Book: Pedographs and Gait Analysis and Clinical Case Studies
Electronic copies available here:

-Amazon/Kindle:
http://www.amazon.com/Pedographs-Gait-Analysis-Clinical-Studies-ebook/dp/B00AC18M3E

-Barnes and Noble / Nook Reader:
http://www.barnesandnoble.com/w/pedographs-and-gait-analysis-ivo-waerlop-and-shawn-allen/1112754833?ean=9781466953895

https://itunes.apple.com/us/book/pedographs-and-gait-analysis/id554516085?mt=11

-Hardcopy available from our publisher:
http://bookstore.trafford.com/Products/SKU-000155825/Pedographs-and-Gait-Analysis.aspx

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Show Notes:

New device to get people with paralysis back on their feet
Scientists have tested the world’s first minimally-invasive brain-machine interface, designed to control an exoskeleton with the power of thought
https://www.sciencedaily.com/releases/2016/02/160208124241.htm

Splicing out torsions, and aberrant foo types ? Club foot ? etc
http://gizmodo.com/everything-you-need-to-know-about-crispr-the-new-tool-1702114381

Scientists Capture Crispr’s Gene-Cutting in Action
http://www.wired.com/2016/01/crispr-modification/

The UK Just Green-Lit Crispr Gene Editing in Human Embryos
http://www.wired.com/2016/02/the-uk-just-green-lit-crispr-gene-editing-in-human-embryos/

Asymmetries in limbs
http://journals.lww.com/acsm-msse/Abstract/publishahead/Musculoskeletal_Asymmetry_in_Football_Athletes___A.97584.aspx

Tension or compression ?
link to full text: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3676165/

Concept: the forces have to go somewhere, it is a “passing the buck” system.  
We did this blog post here to explain:
http://thegaitguys.tumblr.com/post/138680011664/the-banana-toe-the-force-has-to-go-somewhere

The new muscle discovery !
http://www.rmtedu.com/blog/tensor-vastus-intermedius
http://www.rmtedu.com/blog/tensor-vastus-intermedius
http://www.ncbi.nlm.nih.gov/pubmed/26732825

tendinopathy vasculature: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4650849/

tendinopathy treatment paper: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2505250/

The Great toe’s effect on external hip rotation.

We have a simple video for you today. 

When we assess our clients for gait and locomotion we do a quick screen of all the big player joints, from the toes at least up into the thoracic spine to start. Loss of mobility/range of motion means probable functional impairment. 

In this video we display the effects of the Windlass Mechanism of the great toe. A windlass mechanism according to Wikipedia is:

a type of winch used especially on ships to hoist anchors and haul on mooring lines and, especially formerly, to lower buckets into and hoist them up from wells.

In this case, dorsiflexing the big toe spools the plantarfascia and flexor hallucis longus and brevis around the metatarsophalangeal joint (1st. MTPJ), thus pulling the heel towards the forefoot thus raising the arch. When the arch raises, the talus moves cephalad (upwards) and because of the supinatory movement orientation, it spins the tibial externally which in turn spins the femur externally. This is what you see in this video, note the blue dots being carried laterally with the limb external rotation.

The point here today, if you have loss of external hip rotation, it could be crying for you to evaluate the range of motion of the 1st MTP joint , it could be crying for you to evaluate the skill of toe extension, strength or endurance or all of the above. Impairment of the 1st MTP has great inroads into ineffective locomotion. You must have decent range of motion to effectively supinate, to effectively toe off, to externally rotate the limb, to effectively acquire hip extension to maximize gluteal use.  Thus, one could easily say that impaired hallux/great toe extension (skill, ability, endurance, strength) can impair hip extension (and clean hip extension patterning) and result in possible terminal propulsive gait extension occurring through the lumbar spine instead of through the hip joint proper.

Think of the effects of two asymmetrical great toe extensions, comparing the great toe left to right. Asymmetry in the limbs, pelvis, hip extension and perhaps worse, the lumbar spine, is a virtual guarantee.  Compare hallux extension side to side, if you can achieve symmetry through skill, endurance and strength retraining, you must do it. If you have a hallux limitus, a bunion or anything that impairs the symmetry of great toe extension side to side, you have some interesting work to do. 

You have to know what you have in your client, and know what it means to their locomotion.  Seeing or recognizing what you have must translate into understanding and action. 

Play mental games with clinical entities.  In this case, if at terminal toe off you did not have full hallux extension like in this client, and thus you did not get that last little final external rotation spin in the limb at the hip … . . what could that do to your gait ? Go tape your toe and limit terminal extension (terminal dorsiflexion) and walk around, to feel it in yourself is to get first hand experience. 

Shawn Allen, one of the gait guys

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Pain at toe-off; Stopping Big Toe Impingement with the extensor hallucis capsularis.

Photo: note the AET coming off the EHL tendon in the diagram

What if there was a mechanism in place by which to pull structures out of the way of a joint moving to end range ? If you know your biomechanics, you know this is a true phenomenon on several levels. We know of one at the knee, the articularis genu has been written about having function of drawing the suprapatellar bursa and joint capsule/synovial tissue cephalad (upward) during knee extension preventing an impingement phenomenon during full quadriceps contraction in knee extension loading. 

What if there were a similar mechanism in the big toe ? When teaching we are sometimes asked what joint, that when it goes sour, creates more devastation to the entire biomechanical chain than any other joint. I like to choose the big toe/1st metatarsophalangeal joint because failure to fully push off the big toe at full joint range impairs hip extension, stride and step lengths, and creates compensations far and wide ipsilaterally and contralaterally in the body. Most everyone knows about bunions, turf toe, hallux valgus, sesamoiditis and the like, but there are many other things that can make this joint painful. Today we bring you another “clearing mechanism” that acts to pull synovial and capsular tissues out of a joint that is nearing end range.
As seen in the anatomy dissection photo above, the extensor hallucis capsularis (EHC) is an accessory tendon slip off of the extensor hallucis longus (EHL). Interestingly, one study found that 8% of the dissections showed the EHC came off of the tibialis anterior tendon slip. This EHC accessory slip typically originates off the long extensor tendon (EHL) and traverses medially to the dorsomedial joint capsule region. Some studies suggest it is found in 80-98% of people. We propose it is most likely present in everyone because of the critical nature of its function. We propose that perhaps it may be missed on traditional dissections because of its blending with fascial tissues and because of its sometimes trivial size and girth. Just like when we fully extend our knee we want to be sure the articularis genu will draw the synovial capsular tissue up and out of the patellar/femoral approximation, the EHC has been shown on intra-operative testing to exert a pretension on the metatarsophalangeal (MTP) joint capsule similarly pulling the synovial-capsular tissue free from the end range dorsiflexing toe. Without this function, synovial-capsular impingement can occur and create pain and an inhibitory arthrogenic reflex to the EHL, tibialis anterior or any other muscles around the joint for that matter. This can act and feel like an acute “turf toe” (hyper-dorsiflexion event) and yet, not be true turf toe osseous impingement.
So if your client has pain at the dorsal joint on end range extension of the great toe, meaning things like toe-off, doing push ups from the ball of the foot, jumping, kneeling or squatting with the hallux in forced dorsiflexion etc, this tendon slip (and its origin, the EHL muscle) should be on your mind and assessment of the anterior compartment for S.E.S. must commence (S.E.S.= skill, endurance and strength, our Gait Guys mantra). This is why you need to intimately understand this important video (link) and need to know how to do this exercise, the shuffle walks (video link) and build clean ankle rocker ranges of motion via S.E.S. of the anterior compartment.  Pulling on the great toe, twisting it like a radio knob, and forcing end range shouldn’t be the biggest guns in your arsenal, logically restoring all the dysfunctional components should be.

We wonder how many of the videos online of people demonstrating big toe mobilizations, toe distractions, fancy exercises and various toe circus tricks to regain motion and function and reduce pain actually truly know about the anatomy and function of the big toe and how ankle rocker and other things can impair its function.  We wonder about these kinds of things.  

Please just remember, the average uneducated viewer is merely looking for solutions to their painful parts. Those in the know have a responsibility to deliver as complete a package as possible, within reason. 

“With great powers (and knowledge) there must also come, great responsibility.”-Stan Lee  

Dr. Shawn Allen

the gait guys

Photo credit link: http://www.wisconsinfootandankleinstitute.com

www.wisconsinfootandankleinstitute.com/img/research/The-Accessory-Extensor-Tendon_fig1.jpg

references:

Foot Ankle Surg. 2014 Sep;20(3):192-4. doi: 10.1016/j.fas.2014.04.001. Epub 2014 Apr 16.
The extensor hallucis capsularis tendon–a prospective study of its occurrence and function.Bayer T1, Kolodziejski N2, Flueckiger G2.

Foot Ankle Int. 2006 Mar;27(3):181-4.
Extensor hallucis capsularis: frequency and identification on MRI.
Boyd N1, Brock H, Meier A, Miller R, Mlady G, Firoozbakhsh K.

Foot Ankle Int. 2004 Jun;25(6):387-90.
The accessory extensor tendon of the first metatarsophalangeal joint.
Bibbo C1, Arangio G, Patel DV.

Foot Clearance: We don't think about it until we are face down in the mud, and we have all been there.

How many times have you tripped over something so small and insignificant you can barely believe it ? We have all tripped over a small elevation in a cracked sidewalk or a curled up rug corner.  But sometimes we look back and there is no evidence of a culprit, not even a Hobbit or an elf.  How can this happen ?
Minimum foot clearance (MFC) is defined as the minimum vertical distance between the lowest point of the foot of the swing leg and the walking surface during the swing phase of the gait cycle. In other simpler words, the minimum height all parts of the foot need to clear the ground to progress through the swing phase of the limb without contacting the ground. One could justify that getting as close to this minimal amount without catching the foot is most mechanically advantageous.  But, how close to vulnerability are you willing to get ? And as you age, do you even want to enter the danger zone ? Obviously, insufficient clearance is linked to tripping and falling, which is most concerning in the elderly. 
Trips or falls from insufficient foot clearance can be related to insufficient hallux and toe(s) dorsiflexion (extension), ankle dorsiflexion, knee flexion and/or hip flexion, failure to maintain ipsilateral pelvis neutral ( anterior/posterior pelvis posture shifting), even insufficient hip hike generated by the contralateral hip abductors, namely the gluteus medius in most people’s minds. It can also be from an obvious failed concerted effort of all of the above. Note that some of these biomechanical events are sagittal and some are frontal plane.  However, do not ever forget that the swing leg is moving through the axial plane, supported in part by the abdominal wall, starting from a posteriorly obliqued pelvis at swing initiation into an anteriorly obliqued position at terminal swing. We would be remiss as well if we did not ask the reader to consider the “inverted pendulum theory” effect of controlling the dynamically moving torso over the fixed stance phase leg (yes, we could have said “core stability” but that is so flippantly used these days that many lose appreciation for really what is happening dynamically in human locomotion).  If each component is even slightly insufficient, a summation can lead to failed foot clearance.  This is why a total body examination is necessary, every time, and its why the exclusive use of video gait analysis alone will fail every time in finding the culprit(s). 
When we examine people we all tend to look for biomechanical issues unless one grasps the greater global picture of how the body must work as a whole. When one trips we first tend to look for an external source as the cause such as a turned up rug or an object, but there are plentiful internal causes as well. For example, we have this blog post on people tripping on subway stairs.  In this case, there was a change in the perceptual height of the stairs because of a subconscious, learned and engaged sensory-motor behavior of prior steps upward.  However, do not discount direct, peripheral and lower fields of view vision changes or challenges when it comes to trips and falls. Do not forget to consider vestibular components, illumination and gait speed variables as well.  Even the most subtle change in the environment (transitions from tile to carpet, transitions from treadmill to ground walking etc) can cause a trip or fall if it is subtle enough to avoid detection, especially if one is skirting the edge of MFC (minimal foot clearance) already. And, remember this, gait has components of both anticipatory and reactive adjustments, any sensory-motor adaptive changes that impair the speed, calculation and timely integration of these adjustments can change gait behaviors. Sometimes even perceived fall or trip risk in a client can easily slip them into a shorter step/stride length to encourage less single leg stance phase and more double support phase gait. This occurs often in the elderly. This can be met with a reduced minimal foot clearance by design which in itself can increase risk, especially at the moment of transition from a larger step length to a shorter one. Understanding all age-related and non-age related effects on lower limb trajectory variables as described above and only help the clinician become more competent in gait analysis of your client and in understanding the critical variables that are challenging them. 
Many studies indicate that variability and consistency in a motor pattern such as those necessary for foot clearance are huge keys for predictable patterns and injury prevention, and in this case a predictor for trips and falls.  Barrett’s study concluded that “greater MFC variability was observed in older compared to younger adults and older fallers compared to older non-fallers in the majority of studies. Greater MFC variability may contribute to increased risk of trips and associated falls in older compared to young adults and older fallers compared to older non-fallers.”
Once again we outline our mission, to enlighten everyone into the complexities of gait and how gait is all encompassing.  There are so many variables to gait, many of which will never be noted, detected or reflected on a gait analysis and a camera.  Don’t be a minimalist when it comes to evaluating your client’s gait, simply using a treadmill, a camera and some elaborate computer software are not often going to cut the mustard when it really counts.  A knowledgeable and engaged brain are arguably your best gait analysis tools.  
Remember, what you see in someone’s gait is not their problem, it is their adaptive strategy(s).  That is all you are seeing on your camera and computer screen, compensations, not the source of the problem(s).
Shawn and Ivo
the gait guys

References (some of them): 

1. Gait Posture. 2010 Oct;32(4):429-35. doi: 10.1016/j.gaitpost.2010.07.010. Epub 2010 Aug 7.

A systematic review of the effect of ageing and falls history on minimum foot clearance characteristics during level walking. Barrett RS1, Mills PM, Begg RK.

2. Gait Posture. 2007 Feb;25(2):191-8. Epub 2006 May 4. Minimum foot clearance during walking: strategies for the minimisation of trip-related falls. Begg R1, Best R, Dell’Oro L, Taylor S.

3. Clin Biomech (Bristol, Avon). 2011 Nov;26(9):962-8. doi: 10.1016/j.clinbiomech.2011.05.013. Epub 2011 Jun 29. Ageing and limb dominance effects on foot-ground clearance during treadmill and overground walking. Nagano H1, Begg RK, Sparrow WA, Taylor S.

4. Acta Bioeng Biomech. 2014;16(1):3-9. Differences in gait pattern between the elderly and the young during level walking under low illumination. Choi JS, Kang DW, Shin YH, Tack GR.
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Spanking the orthotic: The effects of hallux limitus on the foot’s longitudinal arch.

But the issues do not stop at the arch. If you have been with us long enough, you will have read about the effects of the anterior compartment (namely the tibialis anterior, extensor digitorum and hallucis and peroneus tertius muscles) strength and endurance on the arch.

Here we have a very troubled foot. This foot has undergone numerous procedures, sadly. Today we will not talk about the hallux varus you see here, a virtual unicorn in practice  (and acquired in this case) nor do we want to discuss the phalangeal varus drift. We want to draw your attention to the obvious impairment of the 1st MTP (metatarsophalangeal joint) dorsiflexion range.  You can see the large dorsal crown of osteophytes, a dorsal buttress to any hallux dorsiflexion.  There is under 10 degrees of dorsiflexion here, not even enough worth mentioning.  We have said it many times before, if you lose a range at one joint usually that range has to be accommodated for proximal or distal to the impaired joint. This is a compensation pattern and you can see it here in the hallux joints themselves.

Here you can see that some of the dorsiflexion range has been acquired in the proximal phalangeal joint.  We like to call this “banana toe” when explaining it to patients, it is a highly technical term but you are welcome to borrow it. This occurred because the joint was constantly seeing the limitation of dorsiflexion of the 1st MTP joint and seeing, and accommodating to, the demands of the need for more dorsiflexion at toe off. 

But, here is the kicker. You have likely seen this video of ours on Youtube on how to acquire a foot tripod from using the toe extensors to raise the arch.  Video link here  and here.  Well, in his patient’s case today, they have a limitation of 1st MTP dorsiflexion, so the ability to maximally raise the arch is impaired. The Windlass mechanism is broken; “winding” of the plantar fascia around the !st MTP mechanism is not sufficiently present. Any limitations in toe extension (ie dorsiflexion) or ankle dorsiflexion will mean that :

1. compensations will need to occur

2. The Windlass mechanism is insufficient

3. gait is impaired at distal swing phase and toe off phases

4. the anterior compartment competence will drop (Skill, endurance, strength) and thus injury can be more easily brought to the table.

In this patient’s case, they came in complaining of burning at the top of the foot and stiffness in the anterior ankle mortise area.  This would only come on after a long brisk walk.  If the walk was brisk yet short, no problems. If the walk was long and slow, no problems.  They clearly had an endurance problem and an endurance challenge in the office showed an immediate failure in under 30 seconds (we will try to shoot a quick video so show our little assessment so be patient with us). The point here today is that if there is a joint limitation, there will be a limitation in skill, strength or endurance and very likely a combination of the 3. If you cannot get to a range, then any skill, endurance or strength beyond that limitation will be lost and require a compensation pattern to occur.  This patient’s arch cannot be restored via the methods we describe here on our blog and it cannot be restored by an orthotic. The orthotic will likely further change, likely in a negative manner, the already limited function of the 1st MPJ. In other words, if you raise the arch, you will shorten the plantar fascia and draw the 1st MET  head towards the heel (part of the function of the Windlass mechanism) and by doing this you will plantarflex the big toe … .  but weren’t we praying for an increase in dorsiflexion of the limitus big toe ? ……..yes, exactly !  So use your head  (and spank the orthotic when you see it used in this manner.  ”Bad orthotic, bad orthotic !”)

So think of all of this the next time you see a turf toe / hallux rigidus/ hallux limitus. Rattles your brain huh !?

This is not stuff for the feint of heart. You gotta know your biomechanics.

Shawn and Ivo … .the gait guys

Addendum for clarity:

a Facebook reader asked a question:

From your post: “if you raise the arch, you will shorten the plantar fascia and draw the 1st MET head towards the heel (part of the function of the Windlass mechanism) and by doing this you will plantarflex the big toe … . but weren’t we praying for an increase in dorsiflexion of the limitus big toe ? ” I always thought when the plantar fascia is shortened, it plantar flexes the 1st metatarsal (1st ray) and extends (dorsiflexes) the 1st MTP joint….

Our response:  

We should have been more clear, our apologies dear reader.  Here is what we should have said , ” The plantar fascia is non-contractile, so it does not shorten. We meant conceptually shorten. When in late stance phase, particularly at toe off when the heel has raised and forefoot loading is occurring, the Windlass mechanism around the 1st MET head (as the hallux is dorsiflexing) is drawing the foot into supination and thus the heel towards the forefoot (ie passive arch lift). This action is driving the 1st MET into plantarflexion in the NORMAL foot.  This will NORMALLy help with increasing hallux dorsiflexion. In this case above, there is a rigid 1st MTP  joint.  So this mechanism cannot occur at all. In this case the plantar fascia will over time retract to the only length it does experience. So, if an orthotic is used, it will press up into the fascia and also plantarflex the 1st MET, which will carry the rigid toe into plantar flexion with it, IN THIS CASE.”

How much does your Hallux Extend?

Last week, on Mondays post, we introduced potential areas for power leaks.

The common areas for leaks are:

  • great toe dorsiflexion
  • loss of ankle rocker
  • loss of knee flexion/extension
  • loss of hip extension
  • loss of balance/ proprioception


let’s take a look at a video of the 1st one:

Power leak 1: Great Toe Dorsiflexion

The big toe needs to extend AT LEAST 40 degrees and CLOSER TO 60 degrees for normal walking and running gait. If you do not have that available range of motion, then you will need to “borrow” it from somewhere else.

Common compensations include:

  • externally rotating the foot and coming off the inside of the great toe. this often causes a callus at the medial aspect of the toe. This places the foot in more pronation (plantar flexion, eversion and abduction) so it is a poorer lever.
  • internally rotating the foot and coming off the outside of the foot. This places the foot in more supination ( dorsiflexion, inversion and adduction) and it is therefore a more rigid lever. This often causes tripping or stumbling because of a lack of adequate dorsiflexion of the foot.
  • lifting the foot off the ground and avoiding toe off at terminal stance phase
  • abbreviating the step length to accommodate the amount of available great toe dorsiflexion.

Are YOU losing power? Tune in here for more tips on this series in the coming weeks!

The Gait Guys. Increasing your gait literacy with each and every post.

Some honest movements across the big toe. Things you need to know as a runner or walker.

Lets see how good you are at this gait game at this point. 

In the video above you should see two things: you should see me manually dorsiflexing / extending the big toe / hallux.  I am creating extension through the 1st metatarsal joint (1st MTP). Essentially I am passively engaging the Windlass Mechanism of Hicks.  This mechanism is essentially a biomechanical event that wraps the end of the plantar fascia over the metatarsal head and 1st MTP joint.  So, when a person raises the heel in gait a moment of dorsiflexion/extension occurs across the joint.  In the video you see me lifting the toe but in the closed chain event the toe stays on the ground and foot moves up and over the toe but the resultant motion at the 1st MTP is the same.  It is still dorsiflexion / extension of the big toe about the 1st MTP joint. 
This wrapping or winding of the plantarfascia around the joint causes the distance from the heel to the ball of the foot to shorten and thus creates an elevation of the arch of the foot (smarter than using an orthotic to push it up  huh !) but this mechanism also raises the talus and supinates the foot.  This action makes the foot more rigid and stable. After all, when you are raising the heel and progressing over the ball of the foot don’t you want a rigid lever to press off of ?!  Ask any sprinter and they will concur. However, this mechanism occurs in all folks who  have a relatively competent foot and 1st MTP joint. 
What you NEED to see in the video is the additional motion up the limb. Watch the video again. When the toe is extended (dorsiflexed) the arch rises but the limb also externally rotates. You can see this by the subtle drift of the blue dots on the limb.  We want and need this external rotation to occur at the hip and through the limb because remember, the limb was internally rotated as we passed our body mass over the foot. This is a normal gait phenomenon.
Here is what we want to you to ponder.  Imagine a person with:

  •  a weak extensor hallucis mechanism (both longus and brevis: EHL, EHB)
  •  a hallux rigidus where the toe does not fully extend to that magical 45+ degree range or
  • if the arch of the foot is so far collapsed and pronated

IF any of these things go wrong, then a sufficiently rigid foot is not formed for push off propulsion, an incompetent arch results and insufficient talar motion and external limb rotation will occur. This means that several subsequent biomechanical events will/ may be compromised including but not limited to:

  • contralateral arm swing
  • sufficient engagement of the gluteals for stablization and propusion since optimal external hip rotation will not be met
  • inadequate pelvis posturing for spine neutrality 
  • etc. this is potentially a very very long list

Our take home point here is simple. You must have:

  •  sufficient and relatively symmetrical bilateral big toe extenion (hallux dorsiflexion) to optimize the Windlass Effect
  • you must have sufficient strength of the toe extensors to gain and stabilize this joint range at the 1st MTP joint
  • you must have both of the prior 2 in order to properly posture the foot and arch for rigid terminal loading response
  • and you must have all 3 to sufficiently externally rotate the limb at the hip to engage the propulsive components of the gait cycle.

There is so much more we could have added to this brief blog post, including hallux impingement, hallux valgus and bunion formation, supination insufficiency syndromes, tibialis posterior insufficiency syndrome, metatarsalgia etc.  The list is endless. 

Hallux dorsiflexion, both passive and active range of motion must be checked on every athlete and client you see. Otherwise gait is likely to be impaired locally and globally from subtle insufficiencies.

Shawn and Ivo
The Gait Guys