Whoa! Dangerous shoes ahead....

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Holy smokes ! Can you believe this?

Take a look at these BRAND NEW, just out of the box pair of Brooks Cadence shoes. We do not usually see many manufacturer defects from this brand. Looks like someone might have been asleep at the “upper goes on the midsole” machine

Check out the varus cant to the rearfoot of the right shoe. Now look at the forefoot valgus cant to the left shoe. This would not be a great shoe for someone who has too much rear foot eversion and midfoot pronation on the right and and uncompensated forefoot valgus on the left, but we do not think it was designed for that specific, small niche market.

Think of the biomechanical implications on a "neutral" foot. Placing the right rear foot in varus would effectively halt or slow pronation in the rear foot and midfoot of that foot. This could be a good thing for an over pronator but, in a neutral foot, this would cause them to toe off in supination on that side resulting in low gear push off and biomechanical insufficiency, not to mention the increased external rotation of the lower extremity and lack of shock absorption from 1 of the 4 mechanisms of shock absorption left (mid foot pronation, ankle dorsiflexion, knee flexion, thumb flexion, contralateral drop of the pelvis). Now, imagine if that same person had internal tibial torsion. Talk about placing the knee outside of the sagittal plane ! Can you say macerated meniscus?

And now the left shoe. Look at the valgus cant! If you had and uncompensated forefoot valgus, where the forefoot is everted with respect to the rear foot or a forefoot varus, where they had adequate range of motion to allow the first ray to descend, then this could be a good thing, otherwise they are toeing off in too much pronation. This could be a real problem for a midfoot pronator or someone with large amounts of external tibial torsion, because they commonly toe off in too much pronation and low gear to begin with, as this shoe would accelerate pronation from midfoot to the forefoot

The bottom line? Look at your patients/clients shoes, as well as your own before purchasing them and examined for manufacturer defects. The upper should sit squarely on the midsole and the shoe should not rock or tip from side to side.

TGG

The Sartorius: insertional tendinitis and medial knee pain?

We all see folks with medial knee pain, many times women, with the pain located just below the medial tibial plateau. It often results from running, but sometimes with jumping sports like basketball as well. It has been our experience that these people are often diagnosed with an MCL type injury, but when you examine them further, they do not really fit the bill. All the ligaments are stable and there is no tenderness at the joint line. The is often tenderness at the pes anserine, but who is driving the bus here?

image source: https://commons.wikimedia.org/wiki/File:Muscles_and_tendons_of_the_legs_and_feet;_écorch_́figur_Wellcome_V0008276.jpg

image source: https://commons.wikimedia.org/wiki/File:Muscles_and_tendons_of_the_legs_and_feet;_écorch_́figur_Wellcome_V0008276.jpg

The sartorius originates from the anterior compartment of the thigh. During an ideal gait cycle, the sartorius fires from toe off through nearly terminal swing (1)

We remember that the abdominals should initiate thigh flexion with the iliopsoas, rectus femoris, tensor fascia lata and sartorius perpetuating the motion. Sometimes, when the abdominals are insufficient, we will substitute other thigh flexors, often the psoas and/or rectus femoris, but sometimes sartorius, especially in people with excessive midfoot pronation. Think about all of the medial rotation occurring at the knee during excessive midfoot pronation and when overpronation occurs, the extra compensatory external rotation that must occur to try and bring the knee back into the sagittal plane. The sartorius is positioned perfectly for this function, along with the semitendinosus which assists and external rotation in closed chain. This is why it is often implicated as the culprit in many cases of pes anserine bursitis (or as we like to say “sartorius insertional tendinitis” (2-3)

Some other things you may find interesting is that it is utilized more in crossing or cutting maneuvers while changing directions while running (4). This makes sense, given its anatomical course and origin/insertion. It can often be overlooked in adductor strains. It can also be avulsed during sprints, particularly in adolescents (5) and because of the course of the lateral femoral cutaneus nerve beneath it, can be the cause of meralgia paresthetica (6). It is proprotionally smaller in females (along with the gracilis and short head of the biceps femoris) (7). And during vertical jumping, is considered an internal rotator, along with the semimembranosis, semitendinosis, gracilis, and popliteus (8).

The sartorius is superficial in the anterior thigh, just under the skin, running from the ASIS, coursing lateral to medial and inserting at the pes anserine at its most superior aspect, just overlying the gracilis. Since it is an external rotator, knee flexor and assists in thigh abduction, you can easliy locate it by placing the patient in a "figure 4" position and having them resist as you pull downward on the leg. Be careful if you are needling this muscle because of the subsartorial canal (ie Hunters canal) lying just beneath it in the middle 1/3 of the thigh, from the apex of the femoral triangle to the adductor hiatus in the adductor magnus. It houses the femoral artery and vein, as well as the saphenous nerve and nerve to the vastus medialis.

 

  1. Michaud T: in Human Locomotion: The Conservative Management of Gait-Related Disorders 2011

  2. Imani F, Rahimzadeh P, Abolhasan Gharehdag F, Faiz SH. Sonoanatomic variation of pes anserine bursa. Korean J Pain. 2013;26(3):249-54. 

  3. Gupta, Aman & Saraf, Abhinesh & Yadav, Chandrajeet. (2013). ISSN 2347-954X (Print) High-Resolution Ultrasonography in PesAnserinus Bursitis: Case Report and Literature Review. 1. 753-757. 

  4. Rand MK, Ohtsuki T. EMG analysis of lower limb muscles in humans during quick change in running directions. Gait Posture. 2000 Oct;12(2):169-83.

  5. Manning CJ, Singhai S, Marshall P. Synchronised sartorius avulsions in adolescent sprinter. BMJ Case Rep. 2016 Jul 13;2016.

  6. Hsu CY, Wu CM, Lin SW, Cheng KL. Anterior superior iliac spine avulsion fracture presenting as meralgia paraesthetica in an adolescent sprinter. J Rehabil Med. 2014 Feb;46(2):188-90. doi: 10.2340/16501977-1247.

  7. Behan FP, Maden-Wilkinson TM, Pain MTG, Folland JP. Sex differences in muscle morphology of the knee flexors and knee extensors. PLoS One. 2018 Jan 23;13(1):e0190903.

  8. Cleather DJ. An important role of the biarticular hamstrings is to exert internal/external rotation moments on the tibia during vertical jumping. J Theor Biol. 2018 Oct 14;455:101-108

The gastroc can causse ankle dorsi and plantarflexion ? Yup. What ?

The gastroc, does it cause ankle dorsiflexion and ankle plantarflexion ? Yup. What ?

You may think you know the answer, the gastrocs are ankle plantarflexors, because that is the easy one we all recognize. But I stew on things when unique cases come in and do not fit the "normal" models and it got me reviewing principles I need to always keep in mind.

Think about it, the gastroc cross the knee, so it causes knee flexion. And when the knee flexes, the proximal tibia is progressing forward in the sagittal plane. Now remember, the foot is on the ground, so the distal tibia is (relatively) fixated in relation to the upper tibia. So, as this proximal top tibial moves forward, because of gastroc contraction, the muscle is actually causing ankle dorsiflexion !

So, it is it important to know your normal gait cycle events ? Yes, Ivo and i harp on that all the time ! One has to know the normal cycles to know when abnormal gait cycles are presenting clues.
So, am I saying that the gastroc are helpers of ankle rocker and ankle dorsiflexion ? Yes, they can be. It is a timing thing. So, we have to again get out of our model of open chain events, and thinking that only the anterior compartment muscles are ankle dorsiflexors. We also have to remember that a bent knee heel raise is not the same as a straight leg (knee extension) heel raise. One can stimulate and assist in ankle dorsiflexion and the other cannot so much. So, in clients with loss of ankle dorsiflexion/ankle rocker should you be assessing the function of the gastroc at the proximal knee, for its effects of dorsiflexion at the ankle ? Yes. Go ahead and try it, bend knee and straight knee heel raises, they are different beasts. This gets more complicated, and i will go into that next week ! I have had some deeper epiphanies i wish to share.
Also, remember, single and biarticular muscles have varied and vast capabilities. Thus it is always vital to consider whole body movements where muscles have abilities to accelerate, decelerate, and control and stablize joints they span, and do not span, via dynamic coupling.
Dr. Allen

Why is that joint range of motion absent? Here are some thoughts.

Photo courtesy of Pixabay.com

Photo courtesy of Pixabay.com

Is this how you think ? It is how we approach puzzles. . . .

Said client has a loss of internal hip rotation (pick any joint for that matter). . . . .

-is the loss of rotation present because they cannot get the rotation range because there is weakness of the internal rotators . . .

- or perhaps external rotators more dominant, combined with the weakness of the internal rotators

-or, is the loss there because of neuro-protective shortness/tightness because the brain feels that the said internal rotation is a vulnerable range (pain, instability), a range where it cannot protect the joint ?

-or, is it a combination of the above? (not to dismiss other processes of course, such as pelvis, knee or foot mechanical issues, OA, pain etc).

If one does not examine a client, how are they supposed to know this all important information?

*What shows up on a functional screen is merely what they are capable of doing/ recruiting/ engaging. It does not tell you why, nor narrow down the causal possibilities. Hence, driving more internal rotation range is silly, driving more strength into the internal rotator is likewise silly. And, merely adding global strength just might provide the overall presentation with more armor, a better coping strategy. Hence, strength first is not always a brilliant solution.

IF all you have is a hammer, everything is going to look like a nail, or you'll at least treat everything like a simple nail.

Whole-body coordination patterns may become partitioned in particular ways as a function of task requirements

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Toddlers actively reorganize their whole body coordination to maintain walking stability while carrying an object. Hsu WH1, Miranda DL2, Chistolini TL3, Goldfield EC4. Gait Posture. 2016 Oct;50:75-81


Today we seem to be going back to dual-tasking again, in this case utilizing the arms as balance assistance devices, amongst their other functions. However, we all know that walking with a hand in a pocket, or carrying something alters our ability to maximize their ballast-like function. Balanced walking involves freely swinging the limbs in pendullar motion. Changes in arm swing will change gait economy and efficiency. We have all run with a water bottle or bag/briefcase and know how that changes the symmetry and fluidity of our gait.

"Whole-body coordination patterns may become partitioned in particular ways as a function of task requirements.”

Today's research piece discusses toddlers and their function as they carry objects.
"children immediately begin to carry objects as soon as they can walk. One possibility for this early skill development is that whole body coordination during walking may be re-organized into loosely coupled collections of body parts, allowing children to use their arms to perform one function, while the legs perform another. Therefore, this study examines: 1) how carrying an object affects the coordination of the arms and legs during walking, and 2) if carrying an object influences stride length and width." -Hsu et al.
In this study of 10 toddlers with 3-12 months of walking experience were recruited to walk barefoot while carrying or not carrying a small toy.

"Stride length, width, speed, and continuous relative phase (CRP) of the hips and of the shoulders were compared between carrying conditions. While both arms and legs demonstrated destabilization and stabilization throughout the gait cycle, the arms showed a reduction in intra-subject coordination variability in response to carrying an object. Carrying an object may modify the function of the arms from swinging for balance to maintaining hold of an object. The observed period-dependent changes of the inter-limb coordination of the hips and of the shoulders also support this interpretation. Overall, these findings support the view that whole-body coordination patterns may become partitioned in particular ways as a function of task requirements." -Hsu et al.

So once again we will say it, if you are coaching the arm swing YOU want, because you do not like what you see in your client, or if you think you are helping your client get more out of their body in terms of speed, power, efficiency or anything of the sort, know that there is a higher, smarter program running the show. And that program in the client’s CNS is smarter than you when it comes to what they need for whole-body coordination pattern generation.

photo credit: courtesy of Pixabay

Skeletal muscle in running.

Footnotes 7 - White and Red.jpg

"skeletal muscle may provide a fundamentally different mechanical function during running than during activities such as swimming,flying, or jumping. These activities require muscle contractions that produce mechanical work at high rates to overcome drag,produce lift, or accelerate the body. Because running requires negligible work against drag and the average mechanical energy of the body is constant over time (at a steady speed on level ground), the demands of support may be met most economically by muscles that produce force while minimizing mechanical work."- Roberts et al


Muscular Force in Running Turkeys:
The Economy of Minimizing Work
Thomas J. Roberts,* Richard L. Marsh, Peter G. Weyand,
C. Richard Taylor

A great paper on Hallux Limits

Don't let the title fool or dissuade you. 

Here is a great paper to support the post earlier this week on hallux limitus. 

Plantar pressure distribution in older people with osteoarthritis of the first metatarsophalangeal joint (hallux limitus/rigidus)

No surprise that the study found folks with osteoarthritis of the the 1st MPJ had greater maximum force and peak pressures under the hallux as well as the lesser toe than controls. 

BUT here is one of the gems from the study: " However, the plantar pressure changes observed in this study can be explained using the concept of high- and low-gear push off described by Bojson-Moller. This model suggests that there are two metatarsal axes through which propulsion may occur; a transverse axis connecting the first to second metatarsal heads, and an oblique axis connecting the second to fifth metatarsal heads. In the presence of normal first MPJ motion, a ‘‘high-gear’’ push- off occurs through the transverse metatarsal axis, resulting in an efficient transfer of bodyweight. In the presence of restricted first MPJ motion, propulsion through the transverse axis is not possible. Subsequently, a ‘‘low-gear’’ push-off occurs through the oblique axis, which subjects the lateral forefoot and toes to increased loading and results in hyperextension of the interphalangeal joint of the hallux prior to toe-off."

Zammit, G. V., Menz, H. B., Munteanu, S. E. and Landorf, K. B. (2008), Plantar pressure distribution in older people with osteoarthritis of the first metatarsophalangeal joint (hallux limitus/rigidus). J. Orthop. Res., 26: 1665–1669. doi:10.1002/jor.20700.    

link to FREE FULL TEXT: http://onlinelibrary.wiley.com/doi/10.1002/jor.20700/epdf

 

When the boot is the cause of your client's problems/pain.

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Asymmetry seems to matter with pathology.

image credit: https://commons.wikimedia.org/wiki/File:PSM_V46_D167_Outer_surface_of_the_human_brain.jpg

image credit: https://commons.wikimedia.org/wiki/File:PSM_V46_D167_Outer_surface_of_the_human_brain.jpg

When you have low back pain, your gait is apt to be asymmetrical

...And that is just what this study showed. It looked at 82 right leg dominant folks with slightly less than 1/2 of them havong low back pain. The folks with lower back pain spent more time on their non dominant leg at the beginning of a gait cycle and on their dominant leg at the end of it. Not surprising that they wanted to find a more stable base or center their COP over the weight bearing foot, especially in light to the fact that the back has such poor cortical representation.

Sung PS, Danial P. A Kinematic Symmetry Index of Gait Patterns Between Older Adults With and Without Low Back Pain. Spine (Phila Pa 1976). 2017 Dec 1;42(23):E1350-E1356. doi: 10.1097/BRS.0000000000002161.

Want to bring out gait pathology? Add something new into the mix...

image source: https://en.wikipedia.org/wiki/Walking

image source: https://en.wikipedia.org/wiki/Walking

We have talked about bringing out compensations and asymmetries in gait patterns by adding a novel stimulus to the exam, like having the client/patient put their hands over their heads, or close their eyes. Here is yet another tool for your toolbox: having the client walk backwards.

Both forward and backward walking share pattern generation control circuits in the brainstem, providing similar (though reversed) kinematic patterns. Backward walking requires different muscle activation sequences which can highlight subtle gait asymmetries, particularly in individuals that have cortical impairment (like the kids with cerebral palsy in this paper) or perhaps people with more subtle cortical impairments, like cerebellar dysafferentation from abnormal joint and muscle mechanoreceptor input and integration. Don’t believe us or what the study says, try it on yourself! It can be a humbling experience : )

In part, the study concludes: “The observed spatiotemporal asymmetry assessments may reflect both impaired supraspinal control and impaired state of the spinal circuitry.”

The next time you are having a difficult time seeing something in an evaluation, or are trying to bring out an asymmetry, in addition to your other tricks, have them walk backwards.

Cappellini G, Sylos-Labini F, MacLellan MJ, Sacco A, Morelli D, Lacquaniti F, Ivanenko Y. Backward walking highlights gait asymmetries in children with cerebral palsy. J Neurophysiol. 2018 Mar 1;119(3):1153-1165. doi: 10.1152/jn.00679.2017. Epub 2017 Dec 20.

Progressions and injuries

Progressions and injuries.
If your client cannot demonstrate a competent foot tripod, and they load eccentrically too long into the tib posterior, peroneus longus, and they load too much through the arch and blow into too much arch splay (loosely/slang meaning beyond reasonable pronation limits) and perhaps they evert the calcaneus a little too much.......

If . . . . if they cannot do these things properly, soundly and in a controlled fashion during a double support (standing on 2 feet) demonstration, through a simple standing knee and hip bend . . . if they cannot control their feet in this simple skill . . . .

Then, how in the heck can they do this standing on one leg,? how are they going to do it in a lunge? or as they step forward onto the foot ?How in the world will they do it walking ?
How in the world will they do it properly, soundly, running ? Squatting? Deadlifting? jumping ? or or or or . . . .

if you are wondering why your client has problems, maybe it is because they are doing things way beyond the pay grade of their foot's (limb's) abilities, skills, endurance, strength etc. If you do the simple stuff wrong, in a cheating corrupt fashion, you a will certainly do it that way when things really matter (running, lifting, playing sports).
Sometimes you have to start at the beginning, at the starting line. If your client is having pain and problems, more load doesn;t necessarily make it right. Proper loading, progressively introduced, might however.

Podcast 141: Deep dive podcast (lots of random gait and running thoughts).

Topics:

strength, asymmetry, gait, thegaitguys, gaitanalysis, achilles, windlass mechanism

Links to find the podcast:

Look for us on iTunes, Google Play, Podbean, PlayerFM and more.

Just Google "the gait guys podcast".

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doctorallen.co

summitchiroandrehab.com

shawnallen.net

Our website is all you need to remember. Everything you want, need and wish for is right there on the site.
Interested in our stuff ? Want to buy some of our lectures or our National Shoe Fit program? Click here (thegaitguys.com or thegaitguys.tumblr.com) and you will come to our websites. In the tabs, you will find tabs for STORE, SEMINARS, BOOK etc. We also lecture every 3rd Wednesday of the month on onlineCE.com. We have an extensive catalogued library of our courses there, you can take them any time for a nominal fee (~$20).

Our podcast is on iTunes and just about every other podcast harbor site, just google "the gait guys podcast", you will find us.

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Topics and links:

A new neuron has been discovered and named. The rosehip cell
https://www.wired.com/story/meet-the-rosehip-cell-a-new-kind-of-neuron/

Strength does matter in our opinion, especially when it is stacked onto asymmetry and dysfunction.
https://www.ncbi.nlm.nih.gov/pubmed/26566993

Am J Sports Med. 2016 Jan;44(1):105-12. doi: 10.1177/0363546515611632. Epub 2015 Nov 13.
Is Hip Abduction Strength Asymmetry Present in Female Runners in the Early Stages of Patellofemoral Pain Syndrome?
Plastaras C1, McCormick Z2, Nguyen C3, Rho M2, Nack SH2, Roth D4, Casey E5, Carneiro K6, Cucchiara A1, Press J2, McLean J7, Caldera F8.

Positive Work Contribution Shifts from Distal to Proximal Joints during a Prolonged Run
SANNO, MAXIMILIAN1,2; WILLWACHER, STEFFEN1,3; EPRO, GASPAR1,4; BRÜGGEMANN, GERT-PETER1,2,3
Medicine & Science in Sports & Exercise: December 2018 - Volume 50 - Issue 12 - p 2507–2517
https://journals.lww.com/acsm-msse/Citation/2018/12000/Positive_Work_Contribution_Shifts_from_Distal_to.16.aspx

Influence of the windlass mechanism on arch-spring mechanics during dynamic foot arch deformation
Lauren Welte, Luke A. Kelly, Glen A. Lichtwark, Michael J. Rainbow
Published 15 August 2018.DOI: 10.1098/rsif.2018.0270
http://rsif.royalsocietypublishing.org/content/15/145/20180270

Med Sci Sports Exerc. 2018 Dec;50(12):2500-2506. doi: 10.1249/MSS.0000000000001710.
Reducing Impact Loading in Runners: A One-Year Follow-up.
Bowser BJ1, Fellin R2, Milner CE3, Pohl MB4, Davis IS5.
https://www.ncbi.nlm.nih.gov/pubmed/29975300/

Achilles tendon structure differs between competitive distance runners and nonrunners despite no clinical signs or symptoms of midsubstance tendinopathy
Todd J. Hullfish,Kenton L. Hagan, Ellen Casey, Josh R. Baxter
09 Aug 2018https://doi.org/10.1152/japplphysiol.00012.2018
https://www.physiology.org/doi/abs/10.1152/japplphysiol.00012.2018

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More subtle clues..LLD's

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This gentleman presented to the office with left-sided knee pain at the medial collateral ligament following a cutting injury, moving from right to left with the left foot planted. As you can see, he has an anatomical leg length discrepancy with tibial and probable femoral length deficiencies on the left side. Can you see the subtle, increased tone of the long flexors of the toes on the left hand side as it evidenced by the increased prominence of the long extensor tendons to a greater degree on the shorter side? This is a common compensation seen in true leg length discrepancies with clawing of the toes in attempt to create stability on the shorter leg side. Often times, the progression angle on the shorter side will be increased as well.

Pincer toe nails.

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Pincer Toe nails: You've seen them; did you know what they were and how they got that way? Or, did you dismiss them?

We think Hitomi’s hypothesis is correct. Here is why (this is paraphrased from our blog post on subungal hematomas and our revolutionary thinking on why they occur and it seems to fit well with pincer nail formation as well).

… when the skin is pulled at a differential rate over the distal phalange (from gripping of the toes rather than downward pressing through the toe pad) there will be a net lifting response of the nail from its bed as the skin is drawn forward of the backward drawn phalange (there is a NET movement of skin forward thus lifting the nail from its bedding). For an at-home example of this, put your hand AND fingers flat on a table top. Now activate JUST your distal long finger flexors so that only the tip of the fingers are in contact with the table top (there will be a small lifting of the fingers). There should be minimal flexion of the distal fingers at this point. Note the spreading and flattening of the nail. Now, without letting the finger tip-skin contact point move at all from the table, go ahead and increase your long flexor tone/pull fairly aggressively. You are in essence trying to pull the finger backward into flexion while leaving the skin pad in the same place on the table. Feel the pressure building under the distal tip of the finger nail as the skin is RELATIVELY drawn forward.] This is fat pad and skin being drawn forward (relative to the phalange bone being drawn backward) into the apex of the nail. Could this be magnifying the curvature of the nail and not offsetting the “automatic curving and shrinkage” function of the nail ? We think it is quite possible.

We have more to say on this topic, the above is just an excerpt of our blog post. More here, in the link below

https://thegaitguys.tumblr.com/post/127638788139/pincher-nails-who-knew-written-by-dr-shawn?fbclid=IwAR06ol516n9WF2Qh5TadlKd8esXrH5pVviycT_7QiMeScL0UJ3H9r1FF_OQ

Do you have dorsal (top) foot pain? Think you are tying your shoes too tightly?

Do you have dorsal (top) foot pain, at the peak of the arch? Think you are tying your shoes too tightly and that is the cause? Do you have pain over the dorsal or plantar mid foot on heel rise or jumping/landing or going up stairs ?

Just because you raise your heel and load the ball of the foot does not necessarily mean you have adequately plantarflexed the 1st metatarsal and loaded it soundly/stable with the medial tarsal bone. Heel rise, and thus loading onto the medial foot tripod, must be met with ample, stable, durable, 1st metatarsal plantarflexion and the associated medial tarsal bones. Also, without this, loading of the sesamoids properly cannot occur, and pain may ensue.

The first ray complex can be delicate in people who are symptomatic. In some people who do not have a good tibialis posterior-peroneus sling mechanism working harmoniously, in conjunction with a competent arch tripod complex to achieve a compentent arch complex (ie, EDL, EHL, tib anterior and some of the other foot intrinsics) this tarsometatarsal interval can become painful and instead of the 1st ray complex being stable and plantarflexing as the heel departs and the 1st ray begins taking load, it may not do so in a stable plantarflexed posturing. In some people it can momentarily dorsiflex as the arch subtly collapses (when it should be stable and supinated in heel rise).

"Subtle hypermobility of the first tarsometatarsal joint can occur concomitantly with other pathologies and may be difficult to diagnose. Peroneus Longus muscle might influence stability of this joint. Collapse of the medial longitudinal arch is common in flatfoot deformity and the muscle might also play a role in correcting Meary's angle."-Duallert et al

Soon, I hope to show you a video of how to watch for this problem, how to train it properly, how we do it in my office.
Dr. Allen

https://www.ncbi.nlm.nih.gov/pubmed/27015031

Clin Biomech (Bristol, Avon). 2016 May;34:7-11. doi: 10.1016/j.clinbiomech.2016.03.001. Epub 2016 Mar 10.

The influence of the Peroneus Longus muscle on the foot under axial loading: A CT evaluated dynamic cadaveric model study.

Dullaert K1, Hagen J2, Klos K3, Gueorguiev B4, Lenz M5, Richards RG6, Simons P7.

Your center of mass in relation to foot strike position.

For those arm swing/pulsers/ COM and head over foot folks consider some more research below.
Let the CNS drive the show, it is what it is there for . . . The leg motor patterns are dominant, the arms are passive and "shape" and influence the leg swing as a balance and ballast effect. As we discuss in an upcoming podcast, to cross the arms in a pumping motion across the midline of the body means one has to have compromised scapular mechanics (mostly protraction) to afford that much humeral adduction. This means we are forcing thoracic rotation as well. This means we are reversing what we know is more true, that "arm motion is driven passively by rotation of the thorax (Pontzer et al., 2009), an idea which is supported by shoulder muscle EMG data" (and not thoracic rotation by arm swing). Why would we try to create more unnatural axial spin through the spine when we are actually trying to move forward in the sagittal plane? Why would we try to force more rotation through the spine when the function of the thoracopelvic canister (ie. the core) is to stabilize rotational /angluar momentum? Hmmmm, things to ponder.

"Previous modelling studies have clearly shown that motion of the arms effectively counterbalances the angular momentum of the lower extremities during running (Hamner & Delp, 2013; Hamner et al., 2010). It has further been suggested that arm motion is driven passively by rotation of the thorax (Pontzer et al., 2009), an idea which is supported by shoulder muscle EMG data, consistent with the shoulders as spring-like linkages (Ballesteros, Buchthal, & Rosenfalck, 1965). Our data are con- sistent with this idea, showing motion of the thorax to be in the opposite direction to that of the swinging leg. Pontzer et al. (2009) also suggested that motion of the thorax is driven passively by motion of the pelvis. However, our data shows that the thorax reaches its peak angular velocity earlier than the pelvis, indicating that thorax motion is not completely passively driven by pelvic movements."

-S.J. Preece et al. / Human Movement Science 45 (2016) 110–118

Fatigue matters. Today's article looks at pre and post exercise fatigue and how, on EMG, our body changes.

Photo credit: pixabay.com

Photo credit: pixabay.com

Even for those of us who do (and should) know better, "the problem is, we are all often knee deep into compensations before we are aware of it, so most of us are always working on adding strength and endurance into our compensations without even knowing it. Our workouts layer things deeper. Yes, almost all of us are on this bus. Don't deny it. The next time you feel that tightness in your shoulder, or in your hip, or feel that tightness or soreness on one side of the low back, or one side of the neck, stop, and ask yourself that honest question. Again, you are on the bus with the rest of us."

We have spent much time discussing our order of things when intervening between a person and what ails them. Namely, our order is to first restore proper skill and patterning, then add endurance (move well often), and then add load, namely strength, power, force, explosive movements and the like. So, Skill, Endurance, Strength. This is a neurologic order, there is good reason for the necessity of this order. We have spend many an hour listening to Dr. Ivo explain why the CNS dictates this is the order with good reason. Cheat this order and you lay down neuroplastic patterns that are anything but what you want for your client. Enough said.

Today we introduce and article that the looks at the lumbo-pelvic-hip complex, a very complicated area, subject to large multi-planar movements and distortions (and hence, large complex multi-planar compensations). We must have good skill, endurance and strength in controlling this massive area safely, meaning, to avoid developing cheating compensatory patterns to negotiate around our days and activities and sports. The problem is, we are often knee deep into compensations before we are aware of it, so most of us are always working on adding strength and endurance into our compensations without even knowing it. Yes, almost all of us are on this bus. Don't deny it. The next time you feel that tightness in your shoulder, or in your hip, or feel that tightness or soreness on one side of the low back, or one side of the neck, stop, and ask yourself that honest question. Again, you are on the bus with the rest of us.

Today's article looks at pre and post exercise fatigue and how, on EMG, our body changes. Now keep in mind, and I will remind you of this again at the end of today's writing, keep in mind of the asymmetries, poor-skill, poor-endurance and poor strength in some areas that pre-exist, before even starting into our exercises. Imagine, assume, that these were there in all of this study's subjects, even prior to the exercise challenge. You should now fully grasp how layered things get for our clients.

Here is what the article said,

"fatigue may affect muscle recruitment, active muscle stiffness and trunk kinematics, compromising trunk stability".-Chang et al.

"The purpose of this study was to compare trunk muscle activation patterns, and trunk and lower extremity kinematics during walking gait before and after exercise. Surface electrodes were placed over the rectus abdominis, external oblique, erector spinae, gluteus medius, vastus lateralis, and vastus medialis of twenty-five healthy indviduals."

"The amplitude increased in the rectus abdominis during loading, midstance , terminal stance, and late swing after exercise. Amplitude also increased during swing phase in the erector spinae, vastus lateralis, and vastus medialis after exercise. There was less trunk and hip rotation from initial contact to midstance after exercise. Neuromuscular fatigue significantly influenced the activation patterns of superficial musculature and kinematics of the lumbo-pelvic-hip complex during walking. Increased muscle activation with decreased movement in a fatigued state may represent an effort to increase trunk stiffness to protect lumbo-pelvic-hip structures from overload."-Chang et al

What we found particularly notable was that they found less trunk and hip rotation from initial contact to midstance after exercise. And that, "neuromuscular fatigue significantly influenced the activation patterns of superficial musculature and kinematics of the lumbo-pelvic-hip complex during walking". As they concluded, increased muscle activation with decreased movement in a fatigued state plausibly indicates an effort to increase trunk stiffness as a protective measure. Translation, a protective compensation.

Here is what we have to say about that: do not leave the problem on the table and merely train your client around this. Resolve the underlying problem. The underlying problem may not, and likely will not, come out in a "functional screen". What will come out in the screen is how they are moving about with this existing compensation pattern(s). The screen shows WHAT they are doing with their limitations, not WHY Dive keep dear brethren. This is what it is all about, taking the time and diving deep. Find the "why".

So, as promised, here I am again, reminding you to keep in mind of the asymmetries, poor-skill, poor-endurance and poor strength in some areas that pre-exist, before even starting into our exercises. Imagine, assume, that these were there in all of this study's subjects, even prior to the exercise challenge. You should now fully grasp how layered things get for our clients.This is what can make, "helping someone get well", a difficult challenge, even on a good day.

*Muscle activation patterns of the lumbo-pelvic-hip complex during walking gait before and after exercise. Chang M1, Slater LV2, Corbett RO1, Hart JM1, Hertel J1.

Photo credit: pixabay.com Thank you for making such beautiful photos like this available for free use. Gorgeous photography !

How does hallux valgus and bunion formation cause toe hammering?

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Photo: you need to recognize this predictable pattern.

When the hallux begins to become incompetent, from perhaps pain, hallux rigidus, hallux limitus, bunion and in this case hallux valgus with bunion (in this case rotational instability) when this incompetence kicks in, we must find stability elsewhere. One will often, unconsciously, begin to increase the flexor tone and pressure to try and find stability since one cannot get it sufficiently from the hallux anymore.

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Here you see the tenting up of the 2nd toe, from increased long flexor activity (FDL) over time.
And here is the interesting thing you will notice, over time, the 3rd toe will start the same strategy, then the 4th. We see this often. It is not set in stone, but we see it a lot.
Notice it in your clients feet. Teach them why they are getting hammer toes, flexible ones at first, and possibly rigid ones over time. Hammer toes can have many causes, this is just one.
Solution: find a way to help your client re-find better hallux and medial forefoot stability to halt the progression.