Are you following us on our FACEBOOK page ?
every day we try to add extra stuff that will broaden the info, for example, today we added some extra info onto Facebook regarding muscular infarts such as what House has.
If you are not a daily follower of our Facebook page you are missing out on a little extra info.
Just sayin ……
here is the link……. go ahead…….. click the LIKE button…… WE DARE YA !
This is a great video clip (when you click on the youtube link it might not work. Try clicking here ……go ahead and click that link to watch and then come back. Note to listeners…. there is controversy over the lyrics, there always has been and always will be …..but they are listed below at the end of the post.)
When can you ever go wrong with AC/DC ? Combine that with Hugh Laurie from HOUSE MD and you have a great mix.
So, watching this video, why is he using his cane incorrectly? We all know that House’s has a problem with the right hip and leg. “The Rules” state that with a hip problem the cane should always be used on the opposite side to change the D2 lever arm (Click here for a great lesson on this) with a nice follow up here (click). After watching these 2 Gait Guys videos you will clearly understand (perhaps to a better level than most of your therapists and doctors who gave you the cane) why it is used on the opposite side.
So, why in the world is the brilliant Dr. House using it on the same side ? We have received this question more than once. And the answer is quite simple. His problem is extracapsular. In the pilot episode of House MD it was explained that he suffered a vascular infarct to the quadriceps muscle. Like bone infarcts, muscular infarcts can be painful. If he contracts the quadriceps when loading the leg there will be pain. Just like if the infarct were osseous, the loading of the cortical bone and stress on the trabecular infrastructure in that case, axial loading of the limb (muscular or osseous) will drive pain. So, to lessen the issue he uses the cane on the same side to literally share his body mass load over the length of the cane. He is essentially attempting to use the cane as his weight bearing limb. The cane use on the opposite side is best used when you are attempting to unload the muscular compressive forces across the hip (acetabulofemoral) joint. Contraction of the gluteus medius generates the greatest joint compressive loading of all of the hip muscles because of its orientation during gait. Thus, utilizing the cane on the opposite side acts as a hydraulic lift necessitating a shift in body mass closer to the joint and reducing the compressive demands on the gluteus medius muscle.
* Rule breaker: sure, you can still use the cane on the same side to reduce the gluteus medius forces, it is just a bit more awkward. But it can be done. Think about and elderly folk who had a weaker opposite arm, they would feel more comfortable using House’s strategy. The rules are not hard pressed.
So, House is using the cane correctly for his condition.
Rules are meant to be broken. Look at our leaders (all parties) in Washington, they do it everyday ! And when you are as smart as House you know when to break the rules.
Thanks for the reminder AC/DC ……lyrics
“Living easy, living free
Season ticket on a one-way ride
Hey Momma, look at me
I’m on my way to the promised land.
Asking nothing, leave me be
Taking everything in my stride
Dont need reason, dont need rhyme
Aint nothing I’d rather do”
Shawn and Ivo……….. or maybe it is Beavis & Butthead ?
(uh, that’s cool dude ! huhhh huhhhhh …… Those Gait Guys Rule…… !!!! )
chase the article through the linked above
Well, with the Barefoot Running Craze, and all the misinformation out there, we thought you would appreciate this short video
Have a great Friday…
Ivo and Shawn
After a few more questions it was clear that the pain had been around for quite some time and was at a specific pencil eraser sized area above the anterior joint line, slightly medially to center and without question not at the joint line proper but directly on the medial femoral condyle. So, do you know what he has ? You should always suspect this in knees that hyperextend this far or in athlete that have sustained or repetitive hyperextension stressing:
This little fella has the last 2 factors, BIG TIME !
So, clearly understanding these biomechanical factors and coupling a palpatory tenderness at the correct spot on the medial femoral condyle indicates that he has (the youngest we have ever seen)…….drum roll…….
Anterior meniscofemoral impingement syndrome. Never heard of it ? Probably not. Why, because it was glazed over in school, and maybe not at all for many doctors to be honest. Go ahead, look it up under Pubmed and see how many referenced papers you find on it. We see it enough to know that it is frequently diagnosed as a patellar tracking problem but those clients do not have the same risk and anatomy factors. We have had our doctor referrals call us back saying they have never even heard of it. Most have not to be honest. Bottom line, if you know your anatomy and your biomechanics you can figure out most things. If you are slim and skinny on either one you might be missing a few things. We do sometimes as well.
Summary: When the knee hyperextends either too much, too long, or for too many repetitions either statically or in dynamic walking, running or in activity the leading upper edge of the medial meniscus (see anatomy diagram above) can impinge repeatedly and forcefully into the soft medial femoral articular cartilage (see the colored purple area in the diagram) and over time create a softening of the cartilage (condromalacia as it is known). Do it long enough or enough times and you create an inflammatory reaction with a cartilagenous defect.
This poor little guy was hating walking. Interestingly, what do you think happened when we had him crouch walk (knees flexed)……yup…..no pain. He looked up at me in wonderment immediately and of course saw us smiling knowing very well he would be pain free.
Solution in a 4 year old. Slightly flex the knees and place a long strip of tape down the back side of the upper and lower leg. If he extends the knee he forces the tape taught and is instantly reminded (pseudo biofeedback if you will) that he is approaching the danger zone. As this case and many other find, after a few days the skin gets pretty irritated but that is time to take the tape off and let him go back to his old tricks……. trust us, it is only for a few hours until he will figure it out……meaning….. hyperextension is evil ! Teaching this little guy our now famous “Shuffle walks” (to drive ankle dorsiflexion strength in the tibialis anterior and toe extensors in a posture of knee flexion) was on the menu and we turned it into a fun game for him to play with mom and dad.
Anterior Meniscofemoral Impingement Syndrome. Say it 3 times fast with a mouthful of organic chunky peanut butter. We dare ya ! (Sounds like a whimpy frat house hazing technique if you as me.)
Hope you never see it in a little one. if you do, smile and reach for some tape and put on some 70’s music and shuffle to some oldies.
Shawn and Ivo……. yup, orthopedics is also in our soup of letters after our names. But it ain’t the letters that matter, it is what you do with them. Anyone up for Scrabble ?
* Oh, look, we found one journal article ……from 1996 ! Sad.
Plano Orthopedic and Sports Medicine Center, Texas, USA.
The meniscal impingement syndrome consists of three elements: impaction on the anterior medial femoral condyle by the leading edge of the medial meniscus, articular cartilage damage of at least Outerbridge grade 3, and knee hyperextension of at least 5 degrees. This report reviews this condition in a series of seven knees with an average follow-up of 39 months. The time from the onset of symptoms until surgery averaged 45 months. Treatment consisted of a thorough arthroscopic knee evaluation and debridement of the articular cartilage fragmentation and any impinging synovitis. Postoperative rehabilitation includes extension block bracing, hamstring strengthening, and closed-chain exercise. With this regimen, there was improvement in the Tegner scores and a reduction in postoperative knee hyperextension. Identification of this uncommon condition requires a complete evaluation of the medial femoral condyle in patients with knee hyperextension.
________
Neuromechanics Weekly: Third Installment
FEEL THE PAIN: PART 3
The Pain Pathway
Pain is the emotional response to adequate activation of the nociceptive afferent system.
What?
Pain is an emotional response. We feel (or experience) it in the cingulate gyrus (the gyrus right above the corpus callosum, that thing they cut in “One Flew Over the Cukoo’s Nest, see the 1st picture above). Your pain is different than your patients/clients pain. Like John Travolta said in Swordfish "It’s all about perception…”
Pain is subjective. Men usually have lower tolerance for physical pain than women (We think this has to do with their wiring, as it is all connected for them, and we males have our little boxes we keep everything in (see here if you don’t understand).
We know what “adequate activation” is. Enough stimulus to elicit a response. Like when someone keeps pestering you and finally you let them have it!
The “nociceptive afferent system” is the pain pathway. You remember: the C fibers (or pain fibers) in the periphery get activated (adequately, of course), the impulse travels up the peripheral nerve to the dorsal horn of the spinal cord, synapses in lamina 2-5 (the key here is that it synapses; proprioceptive and other sensory stimuli DO NOT synapse, but travel higher up the chain. The synapse allows modulation of the signal, the subject for the next in this series’s post). The next neuron in the pathway (remember, we are still in the cord, right after the 1st synapse) crosses (or decussates) the cord and travels up the ever famous lateral spinothalamic pathway (see middle picture above). We would think this pathway (from the name) goes an synapses at the thalamus next (THE central relay for ALL sensory stimuli EXCEPT SMELL); in reality only about 27% of the fibers synapse here, and then go to the parietal lobe. to tell you WHERE the pain is.
What about the other 73%? They go to the reticular formation ( a loosely organized group of nuclei in the brainstem) to cause the autonomic concomitants of pain (increased heart rate, increased breathing, nausea, urge to urinate, etc).
So, the next time someone has pain in their knee or foot, or _______, not only will you be able to tell what tissue is causing the pain, but now can trace the pathway north to the brain. Why is this important? Because of the modulation that YOU can influence with your therapy. More on that in the next neuromechanics.
The Gait Guys. Eliciting a response in your cingulate gyrus. Hopefully, you are storing this in your inferior temporal gyrus (memory area) for future use.
When the knee hinges sideways. A clinical video case.
This is not a difficult case today, not by any means. Most people will can see what is not normal here. But there are some simple principles we wanted to highlight and remind you of that this case shows nicely.
This is a fairly typical advanced degenerative arthritic right knee and the gait that accompanies it.
Here you can see that when the gentleman steps onto the right limb the knee has a small lateral hinge moment, you can see the knee joint buckle sideways. This is not normal, the knee is supposed to hinge only forward and backwards (flexion and extension) in the sagittal plane. Here it is hinging in the frontal plane. You can easily see that after many years of abnormal stresses that the tibia has deformed into a varus bowed position. This is a great example for you engineer-type out there about long term deformation of solids.
* Deformation of Solids:
- Stress: is a measure of the force required to cause a particular deformation.
- Strain: is a measure of the degree of deformation.
- Elastic Modulus: the ratio of stress to strain:
Elastic modulus = Stress divided by Strain or
EM= Stress / Strain
The lateral forces and hinging over time forced the tibial to varus bow which is a reactionary measure. In simplest of terms, as the bone cells (osteoclasts and osteoblasts) continued to cyclically turn over they laid down new osseous structure along lines of stress which happen to be in the frontal plane, hence the frontal plane bow. At the joint line it was simple to feel and advanced gapping and shifting of the joint in medial-lateral-medial stressing. One can only imagine the maceration of the cartilagenous menisci in such a knee from the abnormal shear forces. Oy !
In this gait, this joint is quite clearly painful as evidenced by the pronounced limp. As right limb weight bearing is initiated carefully and slowly to reduce pain and gain stable purchase of the limb with balance the lateral shift is seen to occur. This lateral shift challenges all of the frontal plane stabilizers so it should be no surprise to anyone that he has significiant gluteus medius, peroneal and abdominal weaknesses in guarding that right frontal plane (to name just a few).
It is most difficult to see on this video because of the loss of 3D specs and because we do not have a frontal view of this gait, but what you typically see in the gait of these clients is a normal left to right step length and an abbreviated right to left. As the brain loads that right limb there is pain and instability sensed by joint and pain receptors. This sparks an early and abrupt departure off of the right limb and hence an abbreviated and shortened right to left step length. This will impart a quick load onto the left leg with an abrupt loading into the left quadriceps. It is not uncommon at all for these clients to develop anterior knee pain syndromes (such as patellofemoral tracking syndromes) or foot problems because of repeated abrupt mid-forefoot loading which drives significant of calf-posterior compartment loading (this will also drive long toe flexor strategies). Also, an abrupt right to left weight bearing shift will generate excessive left lateral (frontal plane) forces thus it is not uncommon to show or develop left hip issues or to see more sustained supination of the left foot. The Peronei can be challenged too to fend off this over-supination that can frequently occur.
* clinical pearl: In our clinics when we see a one sided increase in toe clench and long flexor tone, even when the client lies down, we will once again review gait and look and test for clinical instabilities of stance phase mechanics on the OPPOSITE side of the long toe flexor evidence (in this case there was increased left long toe flexor evidence and early hammer toe formation). This is a huge key, we have just sold a few acres of the farm giving away this pearl. This is one of our goto tricks to find deeper embedded clinical problems. It is not always the case, because the long flexor problem can be local or same sided but you have to at least consider the thought we have proposed.
This is the exact same gait pattern as in a sprained ankle, in fact, same pattern when any part of a limb is painful. As you leave the healthy left foot the brain already knows that right foot impact is going to be painful so a pre-calculation is make to soften the loading and to reduce the loading time, hence the premature limp off the right and onto the left.
It is also important in these cases of significant unilateral bow/varum of the tibia to investigate whether a true leg length discrepancy has developed. It can be a part of the visual limping/lurching gait but it is part of the deformation of the tibia. In this case we ended up using a 3mm sole lift (don’t use a heel lift, why would you just raise the heel ?) to level out his pelvis to decrease the frequent low back pain and tightness that goes with such a gait and also to reduce the step-down drop onto that degenerative knee. In this case, the lift reduced the degree and rate of lateral hinge and thus reduced much of his pain and back discomfort. By bringing the ground up to his foot he thus did not have to step down onto the right limb which accelerates the lateral shift.
* Try it yourself, find a curb on your street and walk along the top surface of the curb with the left foot, stepping down onto the right foot to street level. Do this for a year and you would quickly appreciate what this gentleman was experiencing daily……to a degree of course. The lift on the right would be warmly welcomed !
We were actually able to keep the client very comfortable for almost a year which got him to a time frame that worked for his work and vacation time frame to have the surgery. This is often what a client needs, time. Just time to plan, to prepare mentally for a TKA (Total Knee Arthroplasty (replacement)).
The laterally hinging knee. It is so much more than just a degenerative joint. There is much to be appreciated and learned from pathologic gait patterns.
We are…… Shawn and Ivo…… The Gait Guys ……. center focused but considered by many to be a little off plumb.
A video case of a gait impairment. Chronic dorsal foot pain.
This client came to see us recently. They had a current (2 year) history of dorsal foot achey/burning pain and anterior ankle pain, right greater than left. They had been just about everywhere for these complaints and were pretty much resolved that it was not fixable. They also had a chronic history of anterior shin splints.
This is a pretty simple case. It is missed alot of the time. The reason it is missed is because nothing much shows up on examination. However, we used some tricks to bring out their symptoms. There are also some subtle hints on the gait video above but when you cannot pair what you see with what you find on a clinical exam the issues can get lost in the mix, as they did in this case. This is thus a case based much on clinical experience. We have seen this before. A great clinician (who’s name we have forgotten) used to have a quote that went something like this:
It is only after you have seen the beast once before that it will serve you well to be able to recognize it the next time. Having never seen the beast previously will leave you with a terrible bloody battle on how to slay it the first go-round.“
ln this video above you should basically see 2 things:
1. the easy one to see: the right foot immediately after toe off does not come forward sagitally rather it spins out into abduction in the swing phase to prepare for the next heel strike.
2. the harder one to see: both feet pronate immediately in the rear and mid foot excessively.
This patient has some limitations in normal ankle rocker. More simply put, they cannot get enough adequate tibial progression forward into dorsiflexion over the talar dome. The squat test was really the only positive movement assessment that was confirmatory. As they squatted the ankle met early dorsiflexion restriction and thus the foot had no choice but to pronate early and heavily thus collapsing medially and drawing the knees in medially. Normally the arch should remain unaffected and the tibia should merely pivot cleanly and effortlessly over the talus allowing the knees to come purely forward. Not in this case.
So, we have a client that has impaired sagittal mechanics. They cannot move through ankle rocker effectively and thus they cannot pronate in a timely manner. As the right foot leaves the ground at toe off they need to have sufficient ankle dorsiflexion to carry the foot cleanly forward to prepare for heel strike (this looks pretty good on the left in the video) but the right side is met with ankle range loss. If they did not circumduct the right foot like you see here they would drag their toes on the ground and likely trip. So, foot abduction is the strategy to avoid this issue. However, when you circumduct the foot you begin to lose the strength and endurance of the toe extensors and tibialis anterior.
There is it, we just gave it away. Your question all along should have been, "but what about the dorsal foot and anterior ankle pain and chronic shin splint history?”.
After our gait assessment and history we had a strong hunch. We did our clinical exam which was unremarkable, mostly. But we saw some things that might correlate with our hunches. So, we put the client on our Total Gym at 45 degree incline to do some partially weight bearing squats. Simple stuff. But, we put the feet in a challenged position. We had them hold a neutral foot position on the platform (zero degree progression ankle), the foot was not allowed to spin. We told them they had to keep the toes up at all times and directed them to not let the arch drop or heel spin (these are all compensations to get around impaired ankle rocker in gait, and we see them in her gait video). It seemed simple to them so they began to squat repeatedly, slowly with good form. At about 2 minutes into the movement challenge there began some burning and achey pain reproduction at the dorsal foot from the toes to the anterior ankle. Then it started up their shins. The knees began to hurt. Their toes began to lose their earlier extension/lift. They then started to avoid the depth of the initial first squats so we made them aware and insisted they challenge the initial ranges. After about another 30 seconds the anterior ankle pain began. Our exam was pretty much done. We went back into the room, their pain had stopped. On the exam table it was clear that they now had more toe extension and ankle dorsiflexion range but had no strength in this new range. You see, they initially tested strong in these ranges, but they were strong only in the limited range available to them. On our exam we felt that the ranges were a bit meager, but for some people that is just their anatomy. But we had to be sure, so we gently drove some of those old lost ranges and our examination was concluded.
So, it turns out that this patient had enough weakness in the tibialis anterior and long toe extensors (EDL) sufficient enough to lose ankle rocker ranges over time. When you lose ankle rocker range you meet resistance early. This means you will begin pronation in the foot earlier than normal and begin one or several compensations:
Chronic fatiguing and weakness of the toe extensors and tibialis anterior are frequent findings in many people. Sometimes they are subtle and you have to tease them out.
Now, remember the initial pain quality ? Achey burning pain. Now, lets review last weeks pain posts.
Remember the Krebs cycle? How about glycolysis? What was one of the end products of glycolysis? Lactic acid. Your ability to recycle it and make it into oxaloacetic acid and stuff it back into the Krebs cycle determines your aerobic capacity. When lactic acid builds up, we get muscular inefficiency due to the drop in pH (initially this helps, but too much of a good thing creates a problem), The result? Burning pain. Burning pain is the burn of glycolysis, or muscular overuse.
Aching/ throbbing pain is that deep, boring pain, like a toothache in a bone. It is the pain of the mesoderm, or what is often called sclerotogenous pain. Aching/Throbbing pain is the pain of connective tissue dysfunction (remember that connective tissue is bone, cartilage and collagenous structures like ligaments and tendons). Throbbing pain can sometimes be vascular in origin, as the connective tissue elements of the vessels (the tunica adventitia to be exact) is stretched (which contains a perineural plexus; think about the pain of a migraine headache).
This client had fatigue weakness. This is a physiologic energy production issue. Thus the BURNING pain in the toe extensor muscles. They also had the chronic achey pain of sclerotogenous referral from connective (mesoderm) tissue challenges.
See how this all comes together ?! Putting the pieces together is not hard once you know what the pieces are supposed to do and what their limitations are. Then you have to listen to them and hear what they are telling you.
This was a case that did not have to go on for 2+ years. This client did not need to suffer and become a shoe and orthotic obsessed fanatic (searching for answers on their own). Their body was screaming for someone to just listen and look at its communications.
We started them with our famous Shuffle Walks to drive toe extension, ankle rocker/tibialis anterior strength and then showed them how to use more of both during normal gait. As with most of the cases like this. We will let them go for 2-3 weeks to improve these SKILL and ENDURANCE components of the movement pattern. We bet this one will take 2-3 visits to resolve. As endurance builds and then as STRENGTH (the last component) builds they will own the changes and be pain free. And then return to then normal shoe shopping habits like the rest of the world.
We are The Gait Guys……..saving humanity from the scourge of gait related pain, one lovely person at a time.
Shawn and Ivo
Project walk for the spinal cord injured. Walking, something we take for granted others dream and pray for again. Watch this inspirational video, and SHARE on your FB page to spread the word of their mission.
Chronic ITB -ITBand tightness in a runner. What is the real issue and solution ?
We get dozens of emails daily, and we try to anonymously post a good case weekly.
A Texas lawyer spent 2011 a little bit busier than most people: He ran 113 marathons. R. Laurence Macon spent much of the year and over 200,000 air miles running in 113 marathons. That bests the current record, and the Guinness World Records is currently reviewing Macon’s accomplishment. At 67, he is ….. click on the link to read the article ….
It is Friday Follies on The Gait Guys and we have something a little different for you. Not something gait related but more movement related. Enjoy the short video.
Guillaume Blanchet spent 382 days riding his bike through the streets of Montreal living what appears to be a normal everyday life on his bike. He dedicates the short film to his father, Yves Blanchet where he first got his love for riding a bike.
Everyday something original and entertaining pops up on the internet and today is no exception as a 3 minute short film titled “THE MAN WHO LIVED ON HIS BIKE” is captivating peoples attention. 
Here’s a quick breakdown of the film-
Original= Absolutely
Creative= Definitely
Quality= Well done
Odd= Very, especially when he’s shaving naked while riding a bike.
Entertaining= Well worth watching the whole 3 minutes
You have heard us talk about crouch gait as a rehabilitative exercise (see another post here). Here is some proof that you are working harder
“The results of this analysis indicate that children walking in crouch gait have less passive skeletal support of body weight and utilize substantially higher muscle forces to walk than unimpaired individuals.”
and
“… during crouch gait, these muscles are active throughout single-limb stance, in contrast to the modulation of muscle forces seen during single-limb stance in an unimpaired gait.”
...and working the right muscles
“Crouch gait relies on the same muscles as unimpaired gait to accelerate the mass center upward, including the soleus, vasti, gastrocnemius, gluteus medius, rectus femoris, and gluteus maximus.”
and
“Subjects walking in crouch gait rely more on proximal muscles, including the gluteus medius and hamstrings, to accelerate the mass center forward during single-limb stance than subjects with an unimpaired gait.”
Yup, crouched gait gives you more bang for the buck. Try it….You’ll like it!
Yes, we are the Geeks of Gait…. sifting through and synthesizing the research so you don’t have to
Departments of Mechanical Engineering, Clark Center, Stanford University, Stanford, CA 94305-5450, United States. ksteele@stanford.edu
Pathological movement patterns like crouch gait are characterized by abnormal kinematics and muscle activations that alter how muscles support the body weight during walking. Individual muscles are often the target of interventions to improve crouch gait, yet the roles of individual muscles during crouch gait remain unknown. The goal of this study was to examine how muscles contribute to mass center accelerations and joint angular accelerations during single-limb stance in crouch gait, and compare these contributions to unimpaired gait. Subject-specific dynamic simulations were created for ten children who walked in a mild crouch gait and had no previous surgeries. The simulations were analyzed to determine the acceleration of the mass center and angular accelerations of the hip, knee, and ankle generated by individual muscles.
The results of this analysis indicate that children walking in crouch gait have less passive skeletal support of body weight and utilize substantially higher muscle forces to walk than unimpaired individuals.
Crouch gait relies on the same muscles as unimpaired gait to accelerate the mass center upward, including the soleus, vasti, gastrocnemius, gluteus medius, rectus femoris, and gluteus maximus.
However, during crouch gait, these muscles are active throughout single-limb stance, in contrast to the modulation of muscle forces seen during single-limb stance in an unimpaired gait. Subjects walking in crouch gait rely more on proximal muscles, including the gluteus medius and hamstrings, to accelerate the mass center forward during single-limb stance than subjects with an unimpaired gait.
Copyright 2010 Elsevier Ltd. All rights reserved.
Neuromechanics Weekly: Installment 2 (Now aren’t you lucky to have so much neuro in 1 week!)
FEEL THE PAIN: PART 2
The Character of Pain
In today’s post we hope to help you better understand your pain or the pain that someone else describes to you. The character of the pain can tell you much about what tissues are involved and what might be going on behind the scenes. Understanding the anatomy and physiology of the parts is critical. Thus, this post is going to be a little latin/medical word heavy for some of you….. but trust us, if you spend just a few extra minutes championing these words and owning the concepts below you will forever be better at what you do. Or at the very least, better understand your own pain.
In prior post in this series we talked about the pain producing tissues being derived from one of the primordial tissues, the endoderm, ectoderm or mesoderm. And if it is from the mesoderm, from which of the 3 layers of the somite is it originating ? The sclerotome, the dermatome or myotome? (The mesoderm is the middle embryonic germ layer from which connective tissue, muscle, bone, and the urogenital and circulatory systems develop.)
As we discussed yesterday, pain usually has one of four qualities: burning, aching/throbbing, sharp/stabbing, or electric/shooting. Each one tells us something about where it is coming from.
Remember the Krebs cycle? How about glycolysis? What was one of the end products of glycolysis? Lactic acid. Your ability to recycle it and make it into oxaloacetic acid and stuff it back into the Krebs cycle determines your aerobic capacity. When lactic acid builds up, we get muscular inefficiency due to the drop in pH (initially this helps, but too much of a good thing creates a problem), The result? Burning pain. Burning pain is the burn of glycolysis, or muscular overuse.
Aching/ throbbing pain is that deep, boring pain, like a toothache in a bone. It is the pain of the mesoderm, or what is often called sclerotogenous pain. Aching/Throbbing pain is the pain of connective tissue dysfunction (remember that connective tissue is bone, cartilage and collagenous structures like ligaments and tendons). Throbbing pain can sometimes be vascular in origin, as the connective tissue elements of the vessels (the tunica adventitia to be exact) is stretched (which contains a perineural plexus; think about the pain of a migraine headache).
Shooting/electric pain is the pain of the ectoderm. Think about when you hit your ulnar or peroneal nerves and get that “electric shock” sensation. If you ever have had a herniated disc, you know this pain first hand; sharp and shock like. This pain often travels in the distribution of a nerve root or peripheral nerve.
Sharp/ stabbing pain is the pain of acute tissue damage to one of the 3 layers of the somite (the dermatome, sclerotome or myotome). Think of a sprain (sclerotome) or strain (myotome), or the pain of a shingles outbreak (dermatome). Sharp/stabbing pain is the pain of acute tissue damage.
Keep in mind there is often overlap of pain types, which mean that there is more than one tissue crying out for help (the burning pain in the left hip from gluteus medius insufficiency, combined with the dull, achy pain in the medial knee, from poor control of internal rotation of the thigh).
Pay attention to the character of pain, as it often provides clues to the tissue of origin.
The Gait Guys. Explaining it so you can understand it, one pain free stride length at a time.
Ivo and Shawn
Neuromechanics Weekly:
FEEL THE PAIN: PART 1
Pain Producing Tissues
What usually brings people in to see you or us? Sometimes, it is the desire for better performance, but most often it is pain. We see it daily in our offices. You see it daily in yours. In this installment of Neuromechanics, how about we characterize some of what we are seeing?
We like to think of pain as having one of four qualities: burning, aching/throbbing, sharp/stabbing, or electric/shooting. Each one tells us something about where it is coming from. Before we talk about that, we need to assess “What is the pain producing tissue?" To understand this further, we must delve deeper into tissue types.
We may remember from embryology, around the 3rd week of development, the embryo becomes trilaminar (three layers) forming, the endoderm, the mesoderm and the ectoderm. The endoderm becomes most of our organs (called the splanchnotome), the ectoderm becomes the nervous system, and the mesoderm becomes the muscles, ligaments and bones. The mesoderm coalesces and becomes blocks or segments of tissue called somites. These somites have 3 distinct parts: the dermatome, the myotome and the scerotome.
The dermatome becomes the skin, with it’s segmental innervation (the spinal cord level that supplies that area of skin). Think about when someone has an outbreak of shingles, which often follows a spinal nerve root distribution. We often test sensation along both dermatomal distributions (segmental) and peripheral nerve distributions (with contriobutions from many segmental nerves).
The myotome becomes the muscle and the segmental nerves which supply it. Each segmental level usually corresponds to a function (S1 does plantarflexion of the foot, L5 does dorsiflexion of the foot, etc). This is one of the reasons we muscle test, to tell us which segments may be involved in a problem.
The sclerotome becomes the bone, ligament and tendon supplied by one segmental level (ex. C5 does most of the upper humerus, lateral scapula and clavicle and shoulder capsule). It is what causes the pain associated with sprains or fractures. This is the pain of connective tissues (remember, connective tissues connect muscle to bone AND make up the ultrastructure of the muscle itself!) This is one of the most common pains we encounter in a clinical setting.
Knowing the tissue of origin often leads to a more specific diagnosis etiology of why your client/patient (or YOU) are having a problem.
Next time we delve deeper into pain. Until then, we remain, 2 good looking, aging, nerdy bald guys, Ivo and Shawn.
How do you know if your orthotic is working?
Foot orthotics are easy, no? You get casted, it gets built, you put it in your shoe and you’re good to go, right? Wrong!
Orthotics or Orthotic Therapy as we like to call it in our offices, is an ongoing process. If an orthotic is doing it’s job, your foot should change (for the better) and your prescription should become less. At least in an ideal world.
Remember, orthotics are designed to help you adapt to your environment better. Unlike a footbed (which merely creates a level playing field for the foot), they change the biomechanical function of your foot. A lot should go into getting fit for an orthotic, otherwise they can actually cause some of the problems they are purported to fix!
First of all, there should be a comprehensive history of you and whatever is going on, with an inventory of all your past injuries. That appendectomy or laporoscopy which invaded the abdominal wall could be a culprit for future problems. Next you should have a thorough examination of your lower kinetic chain, including the feet, ankles, knees, hips and low back. This should include range of motion, muscle strength, muscle recruitment patterns and joint function, along with reflexes, sensation and balance or proprioception. Next there should be an analysis of your gait, preferably with stop motion video which allows you to slow down movements and assess subtle abnormalities that may not be visible during normal speeds of movement.
At this point, it should be obvious to both you and your orthotic provider whether or not an orthotic is needed. If so, a non weight bearing cast (weight bearing casts show you what the problem or compensation is, why would you want to cast someone in their pathology and perpetuate it?) The non weight bearing cast is usually done in a mid to terminal stance position of the foot. This should be followed by the prescription of appropriate stretches and exercises, specific to your condition. Shoe recommendations should also be given, since different foot types require different footwear characteristics (good news for the ladies who like shoes!).
So, if you need an orthotic (remember, the prescription should become less over time AND should be accompanied by appropriate exercises), these are the steps we feel are imperative, otherwise, you may just have a really expensive doorstop….
The Gait Guys….Promoting Gait Literacy, one stride length at a time.
Our Response:
Thanks for the kudos. Textured insoles are probably a good thing, as they can help provide more proprioception from the feet (which have a tremendous amount of cortical representation). They could conceivably affect hip and abdominal function. The research out there seems to be focused on people with MS, but here is another supporting study: http:// www.ncbi.nlm.nih.gov/pubmed/18246902
The Gait Guys…Texturing your mind with new ideas….
Gait Issues: When Proprioception is Lost … What we lose when we wear “the wrong” shoes …
You have heard us use this word proprioception a million times (OK, some, maybe not a million). Proprioception is our ability to be aware of and orient our body or a body part in space. Poor proprioception can result in balance and coordination difficulties as well as being a risk factor for injury (Like this poor pooch). *Note: there is no such thing as a “proprioceptor”. All receptors have a more specific name but there are no receptors in the body actually called a proprioceptor, it is a rough classification if that.
Think about people with syphilis, who lose all afferent (sensory) information coming in through the dorsal root ganglia at the spine level. This ultimately leads to a wide based ataxic gait (due to a loss of position and tactile sense) and joint destruction (due to loss of position sense and lack of pain perception). The same consequences can occur, albeit on a smaller scale, when we have diminished proprioception from a joint or its associated muscle spindles. Just like when we put shoes on our feet, proprioception is lost. Just as it would be lost if we wore oven mitts on our hands all day long; there is a cost to optimal functioning of those muted joints.
To review, proprioception is subserved by both cutaneous receptors in the skin (pacinian coprpuscles, Ruffini endings, etc.), joint mechanoreceptors (types I,II,III and IV) and from muscle spindles (nuclear bag and nuclear chain fibers) . It is both conscious and unconscious and travels in two main pathways in the nervous system.
Conscious proprioception arises from the peripheral mechanoreceptors in the skin and joints and travels in the dorsal column system to ultimately end in the thalamus, where the information is relayed to the cortex. Unconscious proprioception arises from joint mechanoreceptors and muscle spindles and travels in the spino-cerebellr pathways to end in the midline vermis and flocculonodular lobe of the cerebellum.
Conscious proprioceptive information is relayed to other areas of the cortex and the cerebellum. Unconscious proprioceptive information is relayed from the cerebellum to the red nucleus to the thalamus and back to the cortex, to get integrated with the conscious proprioceptive information. This information is then sent down the spinal cord to effect some response in the periphery. There is a constant feed back loop between the proprioceptors, the cerebellum and the cerebral cortex. This is what allows us to be balanced and coordinated in out movements and actions.
Thankfully a fashion trend of wearing oven mitts on our hands has never hit the runways, but in a way we continue to do a similar disservice to our feet wearing shoes. Watch the video again and think about this next time you are contemplating, at the very least, a motion control shoe for yourself or a client. If we all walked like this when we put shoes on we would never have done this disservice of footwear to ourselves long ago.
Ivo and Shawn….Good Looking and Proprioceptively different.
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